High Ca2+ Influx During Traumatic Brain Injury Leads to Caspase-1-Dependent Neuroinflammation and Cell Death.
about
A mouse Model of Focal Vascular Injury Induces Astrocyte Reactivity, Tau Oligomers, and Aberrant Behavior.Angiotensin II Causes Neuronal Damage in Stretch-Injured Neurons: Protective Effects of Losartan, an Angiotensin T1 Receptor Blocker.Adult-onset of mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes (MELAS) syndrome with hypothyroidism and psychiatric disorders.Neurodegeneration and Sensorimotor Deficits in the Mouse Model of Traumatic Brain Injury.Neuroprotective Effects of Platonin, a Therapeutic Immunomodulating Medicine, on Traumatic Brain Injury in Mice after Controlled Cortical Impact.
P2860
High Ca2+ Influx During Traumatic Brain Injury Leads to Caspase-1-Dependent Neuroinflammation and Cell Death.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@en
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@nl
type
label
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@en
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@nl
prefLabel
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@en
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@nl
P2093
P2860
P1476
High Ca2+ Influx During Trauma ...... roinflammation and Cell Death.
@en
P2093
Adriano Andrea Conte
Bryan J Pfister
Mathew Long
P M Abdul-Muneer
Vijayalakshmi Santhakumar
P2860
P2888
P304
P356
10.1007/S12035-016-9949-4
P577
2016-06-11T00:00:00Z