Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
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Desmosomes in acquired diseaseEffects of transport inhibitors on the cellular uptake of carboxylated polystyrene nanoparticles in different cell linesDesmoglein 3 acting as an upstream regulator of Rho GTPases, Rac-1/Cdc42 in the regulation of actin organisation and dynamics.Adducin is required for desmosomal cohesion in keratinocytes.Epithelial microfilament regulators show regional distribution in mouse conjunctiva.Xenopus Kazrin interacts with ARVCF-catenin, spectrin and p190B RhoGAP, and modulates RhoA activity and epithelial integrity.Desmoglein 2 is less important than desmoglein 3 for keratinocyte cohesion.The extent of desmoglein 3 depletion in pemphigus vulgaris is dependent on Ca(2+)-induced differentiation: a role in suprabasal epidermal skin splitting?Peptide-mediated desmoglein 3 crosslinking prevents pemphigus vulgaris autoantibody-induced skin blisteringKeratinocyte cytoskeletal roles in cell sheet engineeringDesmosomal cadherins and signaling: lessons from autoimmune disease.Desmosomes and extradesmosomal adhesive signaling contacts in pemphigus.Desmoglein 3: a help or a hindrance in cancer progression?E-cadherin and Src associate with extradesmosomal Dsg3 and modulate desmosome assembly and adhesion.Desmoglein 3 promotes cancer cell migration and invasion by regulating activator protein 1 and protein kinase C-dependent-Ezrin activation.Genome-wide expression analysis suggests unique disease-promoting and disease-preventing signatures in Pemphigus vulgaris.Desmosomes in the testis: Moving into an unchartered territory.Desmosome disassembly in response to pemphigus vulgaris IgG occurs in distinct phases and can be reversed by expression of exogenous Dsg3Inhibition of FAK prevents blister formation in the neonatal mouse model of pemphigus vulgaris.Atomic Force Microscopy Provides New Mechanistic Insights into the Pathogenesis of Pemphigus.Neural nitric oxide synthase participates in pemphigus vulgaris acantholysis through upregulation of Rous sarcoma, mammalian target of rapamycin and focal adhesion kinase.Autoantibody Signaling in Pemphigus Vulgaris: Development of an Integrated Model.Enteropathogenic Escherichia coli EspH-Mediated Rho GTPase Inhibition Results in Desmosomal Perturbations.
P2860
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P2860
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@en
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@nl
type
label
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@en
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@nl
prefLabel
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@en
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@nl
P2093
P2860
P1476
Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.
@en
P2093
Gregory S Harms
Martin Gliem
Volker Spindler
Wolfgang-Moritz Heupel
P2860
P304
P356
10.1152/AJPCELL.00075.2010
P50
P577
2010-06-16T00:00:00Z