E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
about
Functional Roles of E6 and E7 Oncoproteins in HPV-Induced Malignancies at Diverse Anatomical SitesRabGEF1 regulates stem cell factor/c-Kit-mediated signaling events and biological responses in mast cellsp300 alters keratinocyte cell growth and differentiation through regulation of p21(Waf1/CIP1).The human papillomavirus type 58 E7 oncoprotein modulates cell cycle regulatory proteins and abrogates cell cycle checkpoints.Human GTSE-1 regulates p21(CIP1/WAF1) stability conferring resistance to paclitaxel treatment.Retinoblastoma-independent antiproliferative activity of novel intracellular antibodies against the E7 oncoprotein in HPV 16-positive cellsLow- and high-risk human papillomavirus E7 proteins regulate p130 differentlyp63 maintains keratinocyte proliferative capacity through regulation of Skp2-p130 levels.PIK3CA-mediated PI3-kinase signalling is essential for HPV-induced transformation in vitroOncomodulatory signals by regulatory proteins encoded by human cytomegalovirus: a novel role for viral infection in tumor progression.The role of the PI3K/Akt/mTOR signalling pathway in human cancers induced by infection with human papillomaviruses.Human papillomaviruses and cell signaling.Genome-wide analysis of p63 binding sites identifies AP-2 factors as co-regulators of epidermal differentiationRole of Cdk1 in DNA damage-induced G1 checkpoint abrogation by the human papillomavirus E7 oncogene.Increased phosphorylation of p70 S6 kinase is associated with HPV16 infection in cervical cancer and esophageal cancer.Cellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk virusesP21 and p27: roles in carcinogenesis and drug resistance.The papillomavirus E7 proteins.Acetylation regulates the differentiation-specific functions of the retinoblastoma protein.Cooperative regulation of the cell division cycle by the protein kinases RAF and AKTHOPX is methylated and exerts tumour-suppressive function through Ras-induced senescence in human lung cancer.Inactivation of Rb in stromal fibroblasts promotes epithelial cell invasion.The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 protein.Myc and human papillomavirus type 16 E7 genes cooperate to immortalize human keratinocytes.Mitogen-activated protein kinases activate the nuclear localization sequence of human papillomavirus type 11 E1 DNA helicase to promote efficient nuclear import.Myc antagonizes Ras-mediated growth arrest in leukemia cells through the inhibition of the Ras-ERK-p21Cip1 pathway.Human papillomavirus oncoprotein E7 targets the promyelocytic leukemia protein and circumvents cellular senescence via the Rb and p53 tumor suppressor pathways.Chronic NF-kappaB activation delays RasV12-induced premature senescence of human fibroblasts by suppressing the DNA damage checkpoint response.Constitutive activation of the Raf-MAPK pathway causes negative feedback inhibition of Ras-PI3K-AKT and cellular arrest through the EphA2 receptor.
P2860
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P2860
E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年學術文章
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name
E7 abolishes raf-induced arrest via mislocalization of p21
@nl
E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
@en
type
label
E7 abolishes raf-induced arrest via mislocalization of p21
@nl
E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
@en
prefLabel
E7 abolishes raf-induced arrest via mislocalization of p21
@nl
E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
@en
P2093
P2860
P1476
E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).
@en
P2093
Barry R Thrash
Dennis J McCance
Don X Nguyen
Thomas F Westbrook
P2860
P304
P356
10.1128/MCB.22.20.7041-7052.2002
P407
P577
2002-10-01T00:00:00Z