A temporarily distinct subpopulation of slow-cycling melanoma cells is required for continuous tumor growth.
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Melanoma: from melanocyte to genetic alterations and clinical optionsJARID1B is a luminal lineage-driving oncogene in breast cancerAnalysis of the genome to personalize therapy for melanomaA decade of exploring the cancer epigenome - biological and translational implicationsTenascin-C promotes melanoma progression by maintaining the ABCB5-positive side populationPhenotypic plasticity in prostate cancer: role of intrinsically disordered proteinsCancer Stem Cell Quiescence and Plasticity as Major Challenges in Cancer TherapyEvaluating biomarkers to model cancer risk post cosmic ray exposureCancer Stem Cells in the ThyroidTherapeutic strategies targeting cancer stem cellsHistone Modifications, Modifiers and Readers in Melanoma Resistance to Targeted and Immune TherapyTargeting Breast Cancer MetastasisRedox Regulation in Cancer Stem CellsIncreased Oxidative Stress as a Selective Anticancer TherapyCancer stem cells--important players in tumor therapy resistanceThe cancer stem cell niche: how essential is the niche in regulating stemness of tumor cells?Stem cells and targeted approaches to melanoma cureEpigenetic regulation by histone demethylases in hypoxiaReactive oxygen species in cancer stem cellsPathways and therapeutic targets in melanomaMetastatic mechanisms in follicular cell-derived thyroid cancerCan nanomedicines kill cancer stem cells?The "virgin birth", polyploidy, and the origin of cancerKDM5 interacts with Foxo to modulate cellular levels of oxidative stressTumor cells positive and negative for the common cancer stem cell markers are capable of initiating tumor growth and generating both progeniesBet hedging in yeast by heterogeneous, age-correlated expression of a stress protectantEstimating dose painting effects in radiotherapy: a mathematical modelNon-Darwinian dynamics in therapy-induced cancer drug resistance.An inhibitor of KDM5 demethylases reduces survival of drug-tolerant cancer cellsTiming of transcriptional quiescence during gametogenesis is controlled by global histone H3K4 demethylation.Metabolic rewiring in melanomaEpigenetic Therapeutics: A New Weapon in the War Against CancerCyclooxygenase-2: A Role in Cancer Stem Cell Survival and Repopulation of Cancer Cells during TherapyHistone demethylase lysine demethylase 5B in development and cancerCell-of-Origin of Cancer versus Cancer Stem Cells: Assays and InterpretationsAlternative Treatments For Melanoma: Targeting BCL-2 Family Members to De-Bulk and Kill Cancer Stem CellsABCB5 maintains melanoma-initiating cells through a proinflammatory cytokine signaling circuitTGF-β family signaling in stem cellsSenescent cells in growing tumors: population dynamics and cancer stem cellsNonheritable cellular variability accelerates the evolutionary processes of cancer
P2860
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P2860
A temporarily distinct subpopulation of slow-cycling melanoma cells is required for continuous tumor growth.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
A temporarily distinct subpopu ...... d for continuous tumor growth.
@en
A temporarily distinct subpopu ...... d for continuous tumor growth.
@nl
type
label
A temporarily distinct subpopu ...... d for continuous tumor growth.
@en
A temporarily distinct subpopu ...... d for continuous tumor growth.
@nl
prefLabel
A temporarily distinct subpopu ...... d for continuous tumor growth.
@en
A temporarily distinct subpopu ...... d for continuous tumor growth.
@nl
P2093
P2860
P50
P1433
P1476
A temporarily distinct subpopu ...... d for continuous tumor growth.
@en
P2093
Alexander Roesch
Devraj Basu
Elizabeth C Schmidt
Patricia A Brafford
Phyllis Gimotty
Susan E Zabierowski
Thomas Vogt
P2860
P304
P356
10.1016/J.CELL.2010.04.020
P407
P577
2010-05-01T00:00:00Z