Structural analysis of slipped-strand DNA (S-DNA) formed in (CTG)n. (CAG)n repeats from the myotonic dystrophy locus.
about
Molecular basis for expression of common and rare fragile sitesMapping of an origin of DNA replication in the promoter of fragile X gene FMR1Multi-species sequence comparison reveals dynamic evolution of the elastin gene that has involved purifying selection and lineage-specific insertions/deletionsStructural roles of CTG repeats in slippage expansion during DNA replicationMechanistic features of CAG*CTG repeat contractions in cultured cells revealed by a novel genetic assayLength-dependent energetics of (CTG)n and (CAG)n trinucleotide repeats.Role of LrpC from Bacillus subtilis in DNA transactions during DNA repair and recombination.DNA triplet repeat expansion and mismatch repairThe novel double-folded structure of d(GCATGCATGC): a possible model for triplet-repeat sequencesDNA polymorphism in crystals: three stable conformations for the decadeoxynucleotide d(GCATGCATGC)Unusual structures are present in DNA fragments containing super-long Huntingtin CAG repeatsDNA Replication Dynamics of the GGGGCC Repeat of the C9orf72 Gene.Processing of double-R-loops in (CAG)·(CTG) and C9orf72 (GGGGCC)·(GGCCCC) repeats causes instability.Isolated short CTG/CAG DNA slip-outs are repaired efficiently by hMutSbeta, but clustered slip-outs are poorly repaired.Stoichiometry of base excision repair proteins correlates with increased somatic CAG instability in striatum over cerebellum in Huntington's disease transgenic miceInheritance patterns of ATCCT repeat interruptions in spinocerebellar ataxia type 10 (SCA10) expansionsRapid, high fidelity analysis of simple sequence repeats on an electronically active DNA microchip.Topoisomerase 1 and single-strand break repair modulate transcription-induced CAG repeat contraction in human cells.Xpa deficiency reduces CAG trinucleotide repeat instability in neuronal tissues in a mouse model of SCA1.Conformational energetics of stable and metastable states formed by DNA triplet repeat oligonucleotides: implications for triplet expansion diseasesDeterminants of R-loop formation at convergent bidirectionally transcribed trinucleotide repeatsStructure of even/odd trinucleotide repeat sequences modulates persistence of non-B conformations and conversion to duplexDetection of slipped-DNAs at the trinucleotide repeats of the myotonic dystrophy type I disease locus in patient tissues.Global structure of a DNA three-way junction by solution NMR: towards prediction of 3H foldTrinucleotide repeat DNA alters structure to minimize the thermodynamic impact of 8-oxo-7,8-dihydroguanine.Repeat instability during DNA repair: Insights from model systemsAltered replication in human cells promotes DMPK (CTG)(n) · (CAG)(n) repeat instabilityTriptycene-based small molecules modulate (CAG)·(CTG) repeat junctions.Human mismatch repair protein hMutLα is required to repair short slipped-DNAs of trinucleotide repeats.Oligodeoxynucleotide binding to (CTG) · (CAG) microsatellite repeats inhibits replication fork stalling, hairpin formation, and genome instability.Interconverting conformations of slipped-DNA junctions formed by trinucleotide repeats affect repair outcomeRole of DNA secondary structures in fragile site breakage along human chromosome 10A Z-DNA sequence reduces slipped-strand structure formation in the myotonic dystrophy type 2 (CCTG) x (CAGG) repeatInhibition of DNA synthesis facilitates expansion of low-complexity repeats: is strand slippage stimulated by transient local depletion of specific dNTPs?Absence of MutSβ leads to the formation of slipped-DNA for CTG/CAG contractions at primate replication forks.Transcription influences the types of deletion and expansion products in an orientation-dependent manner from GAC*GTC repeatsA brief history of triplet repeat diseases.Abnormal base excision repair at trinucleotide repeats associated with diseases: a tissue-selective mechanism.DNA ligase I competes with FEN1 to expand repetitive DNA sequences in vitro.Transcription-induced DNA toxicity at trinucleotide repeats: double bubble is trouble.
P2860
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P2860
Structural analysis of slipped-strand DNA (S-DNA) formed in (CTG)n. (CAG)n repeats from the myotonic dystrophy locus.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Structural analysis of slipped ...... the myotonic dystrophy locus.
@en
Structural analysis of slipped-strand DNA
@nl
type
label
Structural analysis of slipped ...... the myotonic dystrophy locus.
@en
Structural analysis of slipped-strand DNA
@nl
prefLabel
Structural analysis of slipped ...... the myotonic dystrophy locus.
@en
Structural analysis of slipped-strand DNA
@nl
P2093
P2860
P356
P1476
Structural analysis of slipped ...... the myotonic dystrophy locus.
@en
P2093
C E Pearson
J D Griffith
R R Sinden
P2860
P304
P356
10.1093/NAR/26.3.816
P407
P577
1998-02-01T00:00:00Z