Effects of pharmacological inhibition of NADPH oxidase or iNOS on pro-inflammatory cytokine, palmitic acid or H2O2-induced mouse islet or clonal pancreatic β-cell dysfunction.
about
Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes.Cystine accumulation attenuates insulin release from the pancreatic β-cell due to elevated oxidative stress and decreased ATP levels.Group VIA phospholipase A2 mitigates palmitate-induced β-cell mitochondrial injury and apoptosisControl of Insulin Secretion by Production of Reactive Oxygen Species: Study Performed in Pancreatic Islets from Fed and 48-Hour Fasted Wistar RatsUpregulation of phagocyte-like NADPH oxidase by cytokines in pancreatic beta-cells: attenuation of oxidative and nitrosative stress by 2-bromopalmitate.NADPH oxidase inhibition prevents beta cell dysfunction induced by prolonged elevation of oleate in rodents.Production and function of IL-12 in islets and beta cellsInteraction between cytokines and inflammatory cells in islet dysfunction, insulin resistance and vascular diseaseHigh glucose activates the alternative ACE2/Ang-(1-7)/Mas and APN/Ang IV/IRAP RAS axes in pancreatic β-cells.Omega-3 fatty acids control productions of superoxide and nitrogen oxide and insulin content in INS-1E cells.Bcl-2 maintains the mitochondrial membrane potential, but fails to affect production of reactive oxygen species and endoplasmic reticulum stress, in sodium palmitate-induced β-cell death.Cytotoxicity and cytoprotective effects of citrus flavonoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects.NOX, NOX Who is There? The Contribution of NADPH Oxidase One to Beta Cell Dysfunction.Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line.Inhibition of NADPH oxidase-1 preserves beta cell function.Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion.The role of polyunsaturated fatty acids (n-3 PUFAs) on the pancreatic β-cells and insulin action.
P2860
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P2860
Effects of pharmacological inhibition of NADPH oxidase or iNOS on pro-inflammatory cytokine, palmitic acid or H2O2-induced mouse islet or clonal pancreatic β-cell dysfunction.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@en
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@nl
type
label
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@en
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@nl
prefLabel
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@en
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@nl
P2093
P2860
P356
P1433
P1476
Effects of pharmacological inh ...... pancreatic β-cell dysfunction.
@en
P2093
Gabriele Wolf
Marta Michalska
Reinhard Walther
P2860
P304
P356
10.1042/BSR20090138
P577
2010-12-01T00:00:00Z