about
Lack of functional and expression homology between human and mouse aldo-keto reductase 1C enzymes: implications for modelling human cancersCD8 T cell tolerance to a tumor-associated self-antigen is reversed by CD4 T cells engineered to express the same T cell receptorSelective AKR1C3 inhibitors do not recapitulate the anti-leukaemic activities of the pan-AKR1C inhibitor medroxyprogesterone acetate.Expression of a dominant T-cell receptor can reduce toxicity and enhance tumor protection of allogeneic T-cell therapy.Prostaglandins in muscle regeneration.S-2-hydroxyglutarate regulates CD8+ T-lymphocyte fate.T cell tuning for tumour therapy: Enhancing effector function and memory potential of therapeutic T cells.Genetic Regulation of Fate Decisions in Therapeutic T Cells to Enhance Tumor Protection and Memory Formation.Prostaglandin D2 inhibits C2C12 myogenesis.Memory lapses in graft-versus-host disease.An HIF-1α/VEGF-A Axis in Cytotoxic T Cells Regulates Tumor Progression.Correction: Lack of functional and expression homology between human and mouse aldo-keto reductase 1C enzymes: implications for modelling human cancers.Redirection to the bone marrow improves T cell persistence and antitumor functions.Safety and efficacy of Tet-regulated IL-12 expression in cancer-specific T cells
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description
hulumtues
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wetenschapper
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հետազոտող
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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Pedro Veliça
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P108
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P2798
P31
P496
0000-0002-0557-8544