Smad signaling is required to maintain epigenetic silencing during breast cancer progression. - Wikidata - wikimedia - TriplyDB

Smad signaling is required to maintain epigenetic silencing during breast cancer progression.

Smad signaling is required to maintain epigenetic silencing during breast cancer progression.

http://www.wikidata.org/entity/Q39750283

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DNA methylation changes in genes frequently mutated in sporadic colorectal cancer and in the DNA repair and Wnt/β-catenin signaling pathway genes Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition An insight into the various regulatory mechanisms modulating human DNA methyltransferase 1 stability and function Dysregulation of TGFβ1 Activity in Cancer and Its Influence on the Quality of Anti-Tumor Immunity Role of TGFβ in regulation of the tumor microenvironment and drug delivery (review)TGFβ Signaling in Tumor Initiation, Epithelial-to-Mesenchymal Transition, and Metastasis Genetic variation in the TGF-β signaling pathway and colon and rectal cancer risk.The roles of TGFβ in the tumour microenvironment.Inflammation and Epithelial-Mesenchymal Transition in Pancreatic Ductal Adenocarcinoma: Fighting Against Multiple Opponents HER2 in Breast Cancer Stemness: A Negative Feedback Loop towards Trastuzumab Resistance.Pathway bridge based multiobjective optimization approach for lurking pathway prediction Epigenetic regulation of epithelial-mesenchymal transition Dynamic imbalance between cancer cell subpopulations induced by transforming growth factor beta (TGF-β) is associated with a DNA methylome switch.Identifying global expression patterns and key regulators in epithelial to mesenchymal transition through multi-study integration.Activin-A signaling promotes epithelial-mesenchymal transition, invasion, and metastatic growth of breast cancer Distinct EMT programs control normal mammary stem cells and tumour-initiating cells.Meta-analysis of gene expression signatures defining the epithelial to mesenchymal transition during cancer progression Binding of a Smad4/Ets-1 complex to a novel intragenic regulatory element in exon12 of FPGS underlies decreased gene expression and antifolate resistance in leukemia Multivalent epigenetic marks confer microenvironment-responsive epigenetic plasticity to ovarian cancer cells.Claudins and the modulation of tight junction permeability.Loss of N-Myc interactor promotes epithelial-mesenchymal transition by activation of TGF-β/SMAD signaling.Transforming growth factor-β and the hallmarks of cancer.The high mobility group A2 protein epigenetically silences the Cdh1 gene during epithelial-to-mesenchymal transition Multiple susceptibility loci for radiation-induced mammary tumorigenesis in F2[Dahl S x R]-intercross rats.An autocrine TGF-beta/ZEB/miR-200 signaling network regulates establishment and maintenance of epithelial-mesenchymal transition.Epigenetic changes associated with inflammation in breast cancer patients treated with chemotherapy Protein kinase C-δ inactivation inhibits the proliferation and survival of cancer stem cells in culture and in vivo Breast cancer methylomes establish an epigenomic foundation for metastasis.Roles of STAT3 and ZEB1 proteins in E-cadherin down-regulation and human colorectal cancer epithelial-mesenchymal transition OPCML is frequently methylated in human colorectal cancer and its restored expression reverses EMT via downregulation of smad signaling.Co-expression of parathyroid hormone related protein and TGF-beta in breast cancer predicts poor survival outcome.Targeting IL13Ralpha2 activates STAT6-TP63 pathway to suppress breast cancer lung metastasis Reprogramming during epithelial to mesenchymal transition under the control of TGFβ TGF-β induces global changes in DNA methylation during the epithelial-to-mesenchymal transition in ovarian cancer cells.TGF-β1 epigenetically modifies Thy-1 expression in primary lung fibroblasts The regulation of cell-cell adhesion during epithelial-mesenchymal transition, motility and tumor progression.Key signaling nodes in mammary gland development and cancer: Smad signal integration in epithelial cell plasticity The complexities of TGF-β action during mammary and squamous cell carcinogenesis.Tumor Cell-Derived Periostin Regulates Cytokines That Maintain Breast Cancer Stem Cells.Multiscale modelling of solid tumour growth: the effect of collagen micromechanics.

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P2860

Smad signaling is required to maintain epigenetic silencing during breast cancer progression.

http://www.wikidata.org/entity/Q39750283

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2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年學術文章

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name

Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ing breast cancer progression.

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Smad signaling is required to ...... ring breast cancer progression

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Arthur W Lambert

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Arunthathi Thiagalingam

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Fangming Gao

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Hamid M Abdolmaleky

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Hongjie Pan

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Marc Lenburg

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Sait Ozturk

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Sam Thiagalingam

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Upender Manne

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P2860

Epigenetic gene silencing in cancer – a mechanism for early oncogenic pathway addiction?

Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

Epithelial-mesenchymal transitions in tumour progression

Making sense of latent TGFbeta activation

The MAD-related protein Smad7 associates with the TGFbeta receptor and functions as an antagonist of TGFbeta signaling

Epithelial-mesenchymal transition: at the crossroads of development and tumor metastasis

Smad transcription factors

The role for NES1 serine protease as a novel tumor suppressor

Genes that mediate breast cancer metastasis to lung

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3

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Yuriy O Alekseyev

Panagiotis Papageorgis

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20086175

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