The innate immune molecule, NOD1, regulates direct killing of Helicobacter pylori by antimicrobial peptides.
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Pattern-recognition receptors and gastric cancerStimulation of platelet apoptosis by peptidoglycan from Staphylococcus aureus 113.Helicobacter pylori downregulates expression of human β-defensin 1 in the gastric mucosa in a type IV secretion-dependent fashion.Data driven linear algebraic methods for analysis of molecular pathways: application to disease progression in shock/trauma.Helicobacter pylori exploits cholesterol-rich microdomains for induction of NF-kappaB-dependent responses and peptidoglycan delivery in epithelial cellsNucleotide-oligomerizing domain-1 (NOD1) receptor activation induces pro-inflammatory responses and autophagy in human alveolar macrophagesNOD1-Mediated Mucosal Host Defense against Helicobacter pylori.Vitamin B6 is required for full motility and virulence in Helicobacter pylori.Nod1 and Nod2 signaling does not alter the composition of intestinal bacterial communities at homeostasis.Helicobacter pylori cag pathogenicity island (cagPAI) involved in bacterial internalization and IL-8 induced responses via NOD1- and MyD88-dependent mechanisms in human biliary epithelial cells.H. pylori exploits and manipulates innate and adaptive immune cell signaling pathways to establish persistent infection.Nucleotide Binding Oligomerization Domain 1 Is an Essential Signal Transducer in Human Epithelial Cells Infected with Helicobacter pylori That Induces the Transepithelial Migration of Neutrophils.Activation of type I IFN signaling by NOD1 mediates mucosal host defense against Helicobacter pylori infection.Campylobacter jejuni outer membrane vesicles play an important role in bacterial interactions with human intestinal epithelial cellsCdx2 Expression and Intestinal Metaplasia Induced by H. pylori Infection of Gastric Cells Is Regulated by NOD1-Mediated Innate Immune Responses.The Legionella pneumophila EnhC protein interferes with immunostimulatory muramyl peptide production to evade innate immunityLife in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.Helicobacter pylori CagA: From Pathogenic Mechanisms to Its Use as an Anti-Cancer Vaccine.Role of energy sensor TlpD of Helicobacter pylori in gerbil colonization and genome analyses after adaptation in the gerbil.Detection of enteric pathogens by the nodosome.Role of virulence factors and host cell signaling in the recognition of Helicobacter pylori and the generation of immune responses.Innate immune sensors and gastrointestinal bacterial infections.Nod-like receptors in intestinal homeostasis, inflammation, and cancer.Peptidoglycan: a critical activator of the mammalian immune system during infection and homeostasis.Inflammation, immunity, and vaccine development for Helicobacter pylori.Expression and functional importance of innate immune receptors by intestinal epithelial cells.Peptidoglycan as Nod1 ligand; fragment structures in the environment, chemical synthesis, and their innate immunostimulation.Type I interferons in regulation of mucosal immunity.Role of the Helicobacter pylori-induced inflammatory response in the development of gastric cancer.Differential expression of human beta defensin 2 and 3 in gastric mucosa of Helicobacter pylori-infected individuals.NOD1 and NOD2 Signaling in Infection and Inflammation.Role of Toll-like receptors in Helicobacter pylori infection and immunity.Immune modulation by bacterial outer membrane vesicles.NLR-regulated pathways in cancer: opportunities and obstacles for therapeutic interventions.β-Defensins: Work in Progress.Novel epidermal growth factor receptor pathway mediates release of human β-defensin 3 from Helicobacter pylori-infected gastric epithelial cells.Comparative analysis of the interaction of Helicobacter pylori with human dendritic cells, macrophages, and monocytes.β-Defensins in the Fight against Helicobacter pylori.Antimicrobial peptides in gastrointestinal inflammationHelicobacter pylori induces vascular endothelial growth factor production in gastric epithelial cells through hypoxia-inducible factor-1α-dependent pathway.
P2860
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P2860
The innate immune molecule, NOD1, regulates direct killing of Helicobacter pylori by antimicrobial peptides.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@en
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@nl
type
label
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@en
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@nl
prefLabel
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@en
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@nl
P2093
P2860
P50
P1476
The innate immune molecule, NO ...... ori by antimicrobial peptides.
@en
P2093
Abdulgader Karrar
Cody Allison
Dana J Philpott
Ian A Smith
Jérôme Viala
Luminita Badea
Maria Kaparakis
Shane Reeve
P2860
P304
P356
10.1111/J.1462-5822.2009.01421.X
P577
2009-12-21T00:00:00Z