Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
about
The complex and central role of interferon-γ in graft-versus-host disease and graft-versus-tumor activityCancer-associated fibroblasts in digestive tumorsGene signatures in wound tissue as evidenced by molecular profiling in the chick embryo model.CD248 facilitates tumor growth via its cytoplasmic domain.Tumor expression of CD200 inhibits IL-10 production by tumor-associated myeloid cells and prevents tumor immune evasion of CTL therapy.Adenovirus-mediated delivery of interferon-γ gene inhibits the growth of nasopharyngeal carcinomaPlasticity of γδ T Cells: Impact on the Anti-Tumor ResponseIL-12 suppresses vascular endothelial growth factor receptor 3 expression on tumor vessels by two distinct IFN-gamma-dependent mechanisms.IL-10 contributes to the suppressive function of tumour-associated myeloid cells and enhances myeloid cell accumulation in tumours.Tumor-targeted TNFα stabilizes tumor vessels and enhances active immunotherapy.Multifaceted tumor stromal fibroblastsComplex role of γδ T-cell-derived cytokines and growth factors in cancer.Remodeling of tumor stroma and response to therapy.B-cell lymphoma in a patient with complete interferon gamma receptor 1 deficiencyc-Ski activates cancer-associated fibroblasts to regulate breast cancer cell invasionTNFR2 expression on non-bone marrow-derived cells is crucial for lipopolysaccharide-induced septic shock and downregulation of soluble TNFR2 level in serum.Accelerated tumour metastasis due to interferon-γ receptor-mediated dissociation of perivascular cells from blood vessels.IFNγ-responsiveness of endothelial cells leads to efficient angiostasis in tumours involving down-regulation of Dll4.Interferon-γ produced by tumor-infiltrating NK cells and CD4+ T cells downregulates TNFSF15 expression in vascular endothelial cells.Targeting activation-induced cytidine deaminase overcomes tumor evasion of immunotherapy by CTLs.IFN-γ mediates the antitumor effects of radiation therapy in a murine colon tumor.Tumour ischaemia by interferon-γ resembles physiological blood vessel regression.TNFR-1 on tumor cells contributes to the sensitivity of fibrosarcoma to chemotherapy.Identifying tumor promoting genomic alterations in tumor-associated fibroblasts via retrovirus-insertional mutagenesis.
P2860
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P2860
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@en
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@nl
type
label
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@en
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@nl
prefLabel
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@en
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@nl
P2093
P356
P1476
Responsiveness of stromal fibroblasts to IFN-gamma blocks tumor growth via angiostasis.
@en
P2093
Jingjing Deng
Lianfeng Zhang
Shubai Liu
Zhihai Qin
P304
P356
10.4049/JIMMUNOL.0901073
P407
P577
2009-10-19T00:00:00Z