Leptin and its receptor are overexpressed in brain tumors and correlate with the degree of malignancy. - Wikidata - wikimedia - TriplyDB

Leptin and its receptor are overexpressed in brain tumors and correlate with the degree of malignancy.

Leptin and its receptor are overexpressed in brain tumors and correlate with the degree of malignancy.

http://www.wikidata.org/entity/Q39796091

about

Oncogenic role and therapeutic target of leptin signaling in breast cancer and cancer stem cells Glioblastoma-derived leptin induces tube formation and growth of endothelial cells: comparison with VEGF effects Relationships between hypoxia markers and the leptin system, estrogen receptors in human primary and metastatic breast cancer: effects of preoperative chemotherapy.Design and development of a peptide-based adiponectin receptor agonist for cancer treatment.Leptin signaling molecular actions and drug target in hepatocellular carcinoma Nanoparticles for Targeting Intratumoral Hypoxia: Exploiting a Potential Weakness of Glioblastoma.A dangerous liaison: Leptin and sPLA2-IIA join forces to induce proliferation and migration of astrocytoma cells Metformin inhibits leptin-induced growth and migration of glioblastoma cells.Clinicopathological implications of leptin and leptin receptor expression in papillary thyroid cancer.Correlation of leptin receptor expression with BMI in differential grades of human meningiomas.Leptin's Pro-Angiogenic Signature in Breast Cancer Chronic social isolation is associated with metabolic gene expression changes specific to mammary adipose tissue.High expression of leptin receptor leads to temozolomide resistance with exhibiting stem/progenitor cell features in gliobalastoma.Leptin promotes glioblastoma.pH-sensitive drug-delivery systems for tumor targeting.Oncogenic role and therapeutic target of leptin signaling in colorectal cancer.Adiponectin as novel regulator of cell proliferation in human glioblastoma.Obesity and Cancer Metabolism: A Perspective on Interacting Tumor-Intrinsic and Extrinsic Factors Overexpression of leptin receptor in human glioblastoma: Correlation with vasculogenic mimicry and poor prognosis.Molecular cloning, expression, and regulation of goose leptin receptor gene in adipocytes.Anorexia: an early sign of fourth ventricle astrocytoma in children.Leptin and the brain.

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P2860

Leptin and its receptor are overexpressed in brain tumors and correlate with the degree of malignancy.

http://www.wikidata.org/entity/Q39796091

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2009 nî lūn-bûn

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Leptin and its receptor are ov ...... with the degree of malignancy.

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J.1750-3639.2009.00323.X

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Brain Pathology

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Leptin and its receptor are ov ...... with the degree of malignancy.

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Eva Surmacz

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Gian Luigi Cetto

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Laura Scolaro

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Luis Del Valle

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Marianna Terrasi

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Micol Guidi

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Mirko Riolfi

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Rita Ferla

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Rocco Micciolo

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Sergio Piña-Oviedo

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P2860

The 2007 WHO classification of tumours of the central nervous system

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

Potential role of leptin in angiogenesis: leptin induces endothelial cell proliferation and expression of matrix metalloproteinases in vivo and in vitro

Leptin receptors are developmentally regulated in rat pituitary and hypothalamus

Leptin signaling promotes the growth of mammary tumors and increases the expression of vascular endothelial growth factor (VEGF) and its receptor type two (VEGF-R2)

Malignant astrocytic glioma: genetics, biology, and paths to treatment

Leptin/HER2 crosstalk in breast cancer: in vitro study and preliminary in vivo analysis

Anatomic basis of leptin action in the hypothalamus.

Leptin regulation of proangiogenic molecules in benign and cancerous endometrial cells.

Leptin promotes the proliferative response and invasiveness in human endometrial cancer cells by activating multiple signal-transduction pathways.

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