Successful tumour necrosis factor (TNF) blocking therapy suppresses oxidative stress and hypoxia-induced mitochondrial mutagenesis in inflammatory arthritis.
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Nitric oxide-driven hypoxia initiates synovial angiogenesis, hyperplasia and inflammatory lesions in miceEffect of low-level laser therapy on the expression of inflammatory mediators and on neutrophils and macrophages in acute joint inflammationThe mitochondrial inhibitor oligomycin induces an inflammatory response in the rat knee jointEndothelial dysfunction in chronic inflammatory diseasesChanges in mitochondrial stability during the progression of the Barrett's esophagus disease sequence.Radiological and biochemical effects (CTX-II, MMP-3, 8, and 13) of low-level laser therapy (LLLT) in chronic osteoarthritis in Al-Kharj, Saudi Arabia.Influence of Insulin Resistance and TNF-α on the Inflammatory Process, Oxidative Stress, and Disease Activity in Patients with Rheumatoid Arthritis.4-Hydroxynonenal in the pathogenesis and progression of human diseases.Dysregulated bioenergetics: a key regulator of joint inflammation.Effective tumour necrosis factor-blocking therapy reduces reactive oxygen metabolite level in rheumatoid arthritis.Resolution of TLR2-induced inflammation through manipulation of metabolic pathways in Rheumatoid Arthritis.Oxidative stress as a potential biomarker for determining disease activity in patients with rheumatoid arthritis.Mitochondrial dysfunction promotes and aggravates the inflammatory response in normal human synoviocytes.Human monocytes and macrophages differ in their mechanisms of adaptation to hypoxia.Redox-mediated angiogenesis in the hypoxic joint of inflammatory arthritis.Resveratrol lowers synovial hyperplasia, inflammatory markers and oxidative damage in an acute antigen-induced arthritis model.Oxidative stress impairs energy metabolism in primary cells and synovial tissue of patients with rheumatoid arthritis.Vascular Endothelial Dysfunction in Inflammatory Bowel Diseases: Pharmacological and Nonpharmacological Targets.
P2860
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P2860
Successful tumour necrosis factor (TNF) blocking therapy suppresses oxidative stress and hypoxia-induced mitochondrial mutagenesis in inflammatory arthritis.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Successful tumour necrosis fac ...... sis in inflammatory arthritis.
@en
Successful tumour necrosis factor
@nl
type
label
Successful tumour necrosis fac ...... sis in inflammatory arthritis.
@en
Successful tumour necrosis factor
@nl
prefLabel
Successful tumour necrosis fac ...... sis in inflammatory arthritis.
@en
Successful tumour necrosis factor
@nl
P2093
P2860
P50
P921
P356
P1476
Successful tumour necrosis fac ...... sis in inflammatory arthritis.
@en
P2093
Aisling Kennedy
Douglas J Veale
Emese Balogh
Ting C Chang
P2860
P2888
P356
10.1186/AR3424
P577
2011-07-25T00:00:00Z
P5875
P6179
1003920113