Secretion of Ipa proteins by Shigella flexneri: inducer molecules and kinetics of activation.
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Shigella flexneri LuxS quorum-sensing system modulates virB expression but is not essential for virulenceBacterial type III secretion systems: specialized nanomachines for protein delivery into target cellsStructural and Functional Studies on the N-terminal Domain of the Shigella Type III Secretion Protein MxiGShigella flexneri effectors OspE1 and OspE2 mediate induced adherence to the colonic epithelium following bile salts exposure.Multi-Functional Characteristics of the Pseudomonas aeruginosa Type III Needle-Tip Protein, PcrV; Comparison to Orthologs in other Gram-negative BacteriaSmall-molecule type III secretion system inhibitors block assembly of the Shigella type III secreton.A functional genomic yeast screen to identify pathogenic bacterial proteinsInvolvement of RNA-binding protein Hfq in the osmotic-response regulation of invE gene expression in Shigella sonneiA second wave of Salmonella T3SS1 activity prolongs the lifespan of infected epithelial cells.The Chlamydia trachomatis type III secretion substrates CT142, CT143, and CT144 are secreted into the lumen of the inclusion.The tripartite type III secreton of Shigella flexneri inserts IpaB and IpaC into host membranesMxiM and MxiJ, base elements of the Mxi-Spa type III secretion system of Shigella, interact with and stabilize the MxiD secretin in the cell envelope.Characterization of the promoter, MxiE box and 5' UTR of genes controlled by the activity of the type III secretion apparatus in Shigella flexneriCsrA and Cra influence Shigella flexneri pathogenesis.Spa32 regulates a switch in substrate specificity of the type III secreton of Shigella flexneri from needle components to Ipa proteins.MxiE regulates intracellular expression of factors secreted by the Shigella flexneri 2a type III secretion systemOuter membrane protein A (OmpA): a new player in shigella flexneri protrusion formation and inter-cellular spreading.Chromosomal and plasmid-encoded factors of Shigella flexneri induce secretogenic activity ex vivoOspE2 of Shigella sonnei is required for the maintenance of cell architecture of bacterium-infected cells.Transient suppression of Shigella flexneri type 3 secretion by a protective O-antigen-specific monoclonal IgAOspF and OspC1 are Shigella flexneri type III secretion system effectors that are required for postinvasion aspects of virulenceCharacterization of ExsC and ExsD self-association and heterocomplex formation.Polar localization of PhoN2, a periplasmic virulence-associated factor of Shigella flexneri, is required for proper IcsA exposition at the old bacterial pole.Virulence of enteropathogenic Escherichia coli, a global pathogenEngineering Escherichia coli into a protein delivery system for mammalian cells.Molecular pathogenesis of Shigella spp.: controlling host cell signaling, invasion, and death by type III secretion.The SycN/YscB chaperone-binding domain of YopN is required for the calcium-dependent regulation of Yop secretion by Yersinia pestis.Characterization of SlyA in Shigella flexneri Identifies a Novel Role in VirulenceInduction of type III secretion by cell-free Chlamydia trachomatis elementary bodies.Type III secretion system 1 genes in Vibrio parahaemolyticus are positively regulated by ExsA and negatively regulated by ExsD.Spa15 of Shigella flexneri is secreted through the type III secretion system and prevents staurosporine-induced apoptosis.Identification and discrimination of Burkholderia pseudomallei, B. mallei, and B. thailandensis by real-time PCR targeting type III secretion system genes.Reconstituted human polyclonal plasma-derived secretory-like IgM and IgA maintain the barrier function of epithelial cells infected with an enteropathogen.The synthesis of OspD3 (ShET2) in Shigella flexneri is independent of OspC1.Shigella flexneri IpaH(7.8) facilitates escape of virulent bacteria from the endocytic vacuoles of mouse and human macrophages.Key role for DsbA in cell-to-cell spread of Shigella flexneri, permitting secretion of Ipa proteins into interepithelial protrusions.Inactivation of DsbA, but not DsbC and DsbD, affects the intracellular survival and virulence of Shigella flexneri.Shigella enterotoxin-2 is a type III effector that participates in Shigella-induced interleukin 8 secretion by epithelial cells.Shigella Spa32 is an essential secretory protein for functional type III secretion machinery and uniformity of its needle length.Identification of the cis-acting site involved in activation of promoters regulated by activity of the type III secretion apparatus in Shigella flexneri.
P2860
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P2860
Secretion of Ipa proteins by Shigella flexneri: inducer molecules and kinetics of activation.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@en
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@nl
type
label
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@en
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@nl
prefLabel
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@en
Secretion of Ipa proteins by S ...... es and kinetics of activation.
@nl
P2093
P2860
P1476
Secretion of Ipa proteins by S ...... les and kinetics of activation
@en
P2093
Bahrani FK
Sansonetti PJ
P2860
P304
P407
P577
1997-10-01T00:00:00Z