Human papillomavirus type 16 infection of human keratinocytes requires clathrin and caveolin-1 and is brefeldin a sensitive.
about
Phospholipid scramblase 1 is secreted by a lipid raft-dependent pathway and interacts with the extracellular matrix protein 1 in the dermal epidermal junction zone of human skinHuman papillomavirus infection is inhibited by host autophagy in primary human keratinocytesRiding the R Train into the CellPapillomavirus Infectious Pathways: A Comparison of SystemsConcepts of papillomavirus entry into host cellsEntry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosisDirect binding of retromer to human papillomavirus type 16 minor capsid protein L2 mediates endosome exit during viral infectionRole of Coatomer Protein I in Virus Replication.HPV16 infection of HaCaTs is dependent on β4 integrin, and α6 integrin processing.Human keratinocyte cultures in the investigation of early steps of human papillomavirus infection.Usage of heparan sulfate, integrins, and FAK in HPV16 infection.Vesicular trafficking of incoming human papillomavirus 16 to the Golgi apparatus and endoplasmic reticulum requires γ-secretase activity.Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.The HPV16 and MusPV1 papillomaviruses initially interact with distinct host components on the basement membraneHuman papillomavirus L2 facilitates viral escape from late endosomes via sorting nexin 17.A Dual Role for the Nonreceptor Tyrosine Kinase Pyk2 during the Intracellular Trafficking of Human Papillomavirus 16.Recent developments in the interactions between caveolin and pathogens.Identification of a role for the trans-Golgi network in human papillomavirus 16 pseudovirus infection.Genome-wide siRNA screen identifies the retromer as a cellular entry factor for human papillomavirus.Human papillomavirus types 16, 18, and 31 share similar endocytic requirements for entry.L2, the minor capsid protein of papillomavirus.The role of NH4Cl and cysteine proteases in Human Papillomavirus type 16 infection.Identification of TRAPPC8 as a host factor required for human papillomavirus cell entryThe Role of DCT in HPV16 Infection of HaCaTsOpportunistic intruders: how viruses orchestrate ER functions to infect cells.Host-cell factors involved in papillomavirus entry.Coat as a dagger: the use of capsid proteins to perforate membranes during non-enveloped DNA viruses trafficking.Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus.HPV entry into cells.Smurf2 alters BPV1 trafficking and decreases infection.Alternative autophagy, brefeldin A and viral trafficking pathways.The cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry.
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Human papillomavirus type 16 infection of human keratinocytes requires clathrin and caveolin-1 and is brefeldin a sensitive.
description
2009 nî lūn-bûn
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name
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@en
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@nl
type
label
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@en
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@nl
prefLabel
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@en
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@nl
P2093
P2860
P356
P1433
P1476
Human papillomavirus type 16 i ...... and is brefeldin a sensitive.
@en
P2093
Mallory A Havens
Patricio I Meneses
Sarah A Dabydeen
Valerie Laniosz
P2860
P304
P356
10.1128/JVI.00576-09
P407
P577
2009-06-03T00:00:00Z