Expression of cell cycle proteins in cortical neurons-Correlation with glutamate-induced neurotoxicity.

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Involvement of AMPK in regulating the degradation of MAD2B under high glucose in neuronal cells.

P2860

Q33684371-276A562A-DFC2-434D-9826-322310397002

P2860

Expression of cell cycle proteins in cortical neurons-Correlation with glutamate-induced neurotoxicity.

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http://www.wikidata.org/entity/Q39867433

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

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2016年论文

@zh-cn

name

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

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Expression of cell cycle prote ...... utamate-induced neurotoxicity.

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type

Item

label

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

@en

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

@nl

prefLabel

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

@en

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

@nl

P1476

Expression of cell cycle prote ...... utamate-induced neurotoxicity.

@en

P2093

Arzu Karabay

http://www.w3.org/2001/XMLSchema#string

Yesim Negis

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P2860

Glutamate-induced apoptosis in primary cortical neurons is inhibited by equine estrogens via down-regulation of caspase-3 and prevention of mitochondrial cytochrome c release

Protein kinase C alpha signaling inhibits cyclin D1 translation in intestinal epithelial cells

Calpain activation is required for glutamate-induced p27 down-regulation in cultured cortical neurons

Pin1 inhibition activates cyclin D and produces neurodegenerative pathology

Glutamate-induced apoptosis in neuronal cells is mediated via caspase-dependent and independent mechanisms involving calpain and caspase-3 proteases as well as apoptosis inducing factor (AIF) and this process is inhibited by equine estrogens

Isoform-specific translocation of protein kinase C following glutamate administration in primary hippocampal neurons.

E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3.

Central mammalian neurons normally resistant to glutamate toxicity are made sensitive by elevated extracellular Ca2+: toxicity is blocked by the N-methyl-D-aspartate antagonist MK-801

Cell cycle inhibition provides neuroprotection and reduces glial proliferation and scar formation after traumatic brain injury.

Glutamate receptors, neurotoxicity and neurodegeneration.

P304

358-367

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P31

scholarly article

P356

10.1002/BIOF.1282

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P478

http://www.w3.org/2001/XMLSchema#string

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2016-04-04T00:00:00Z

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P698

27040651

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P921

neurotoxicity

Expression of cell cycle proteins in cortical neurons-Correlation with glutamate-induced neurotoxicity.

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P2860

P2860

Expression of cell cycle proteins in cortical neurons-Correlation with glutamate-induced neurotoxicity.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698

P921

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698

P921