Biophysical characterization of a new SCN5A mutation S1333Y in a SIDS infant linked to long QT syndrome.
about
Multiple arrhythmic syndromes in a newborn, owing to a novel mutation in SCN5ASodium channel biophysics, late sodium current and genetic arrhythmic syndromesCardiac ion channelopathies and the sudden infant death syndromeQRS widening and QT prolongation under bupropion: a unique cardiac electrophysiological profile.Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels.Sudden infant death syndrome and inherited cardiac conditions.
P2860
Biophysical characterization of a new SCN5A mutation S1333Y in a SIDS infant linked to long QT syndrome.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Biophysical characterization o ...... nt linked to long QT syndrome.
@en
Biophysical characterization o ...... nt linked to long QT syndrome.
@nl
type
label
Biophysical characterization o ...... nt linked to long QT syndrome.
@en
Biophysical characterization o ...... nt linked to long QT syndrome.
@nl
prefLabel
Biophysical characterization o ...... nt linked to long QT syndrome.
@en
Biophysical characterization o ...... nt linked to long QT syndrome.
@nl
P2093
P2860
P1433
P1476
Biophysical characterization o ...... nt linked to long QT syndrome.
@en
P2093
Béatrice Kugener
Claire Rodriguez-Lafrasse
Gilles Millat
Mohamed Chahine
Philippe Chevalier
Robert Rousson
P2860
P304
P356
10.1016/J.FEBSLET.2009.02.007
P407
P577
2009-02-10T00:00:00Z