Carbon monoxide rapidly impairs alveolar fluid clearance by inhibiting epithelial sodium channels.
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Carbon monoxide: a critical quantitative analysis and review of the extent and limitations of its second messenger functionReview article: carbon monoxide in gastrointestinal physiology and its potential in therapeuticsEfficacy and safety of inhaled carbon monoxide during pulmonary inflammation in miceHeme oxygenase-1 promotes migration and β-epithelial Na+ channel expression in cytotrophoblasts and ischemic placentasThe carbon monoxide donor, CORM-2, is an antagonist of ATP-gated, human P2X4 receptors.Carbon monoxide: an emerging regulator of ion channels.Alveolar epithelial CNGA1 channels mediate cGMP-stimulated, amiloride-insensitive, lung liquid absorption.Carbon monoxide (CO) is a novel inhibitor of connexin hemichannels.Vocal fold ion transport and mucin expression following acrolein exposure.Therapeutic potential of heme oxygenase-1/carbon monoxide in lung disease.The gasotransmitter hydrogen sulphide decreases Na⁺ transport across pulmonary epithelial cells.AMP-activated protein kinase (AMPK)-dependent and -independent pathways regulate hypoxic inhibition of transepithelial Na+ transport across human airway epithelial cells.TGF-β directs trafficking of the epithelial sodium channel ENaC which has implications for ion and fluid transport in acute lung injury.Ion transport by pulmonary epithelia.Carbon monoxide: an unusual drug.Gasotransmitters: novel regulators of epithelial na(+) transport?Gaseous therapeutics in acute lung injury.Diverse mechanisms underlying the regulation of ion channels by carbon monoxide.Carbon monoxide: A new player in the redox regulation of connexin hemichannels.Mechanistic insight into the heme-independent interplay between iron and carbon monoxide in CFTR and Slo1 BKCa channels.The role of stretch-activated ion channels in acute respiratory distress syndrome: finally a new target?Pharmacological application of carbon monoxide ameliorates islet-directed autoimmunity in mice via anti-inflammatory and anti-apoptotic effects.Biological signaling by carbon monoxide and carbon monoxide-releasing molecules.Nitric oxide inhibits highly selective sodium channels and the Na+/K+-ATPase in H441 cells.Carbon Monoxide Modulates Connexin Function through a Lipid Peroxidation-Dependent Process: A Hypothesis.Amiloride-sensitive sodium channels and pulmonary edema.Carbon Monoxide in Lung Cell Physiology and Disease.Carbon monoxide releasing molecule-2 inhibition of snake venom thrombin-like activity: novel biochemical "brake"?Direct Inhibitory Effects of Carbon Monoxide on Six Venoms Containing Fibrinogenolytic Metalloproteinases.Ion channels as target effectors for carbon monoxideCarbon Monoxide-Releasing Molecule-A1 Inhibits Th1/Th17 and Stimulates Th2 DifferentiationIn vitro
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P2860
Carbon monoxide rapidly impairs alveolar fluid clearance by inhibiting epithelial sodium channels.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
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name
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@en
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@nl
type
label
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@en
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@nl
prefLabel
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@en
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@nl
P2093
P2860
P50
P356
P1476
Carbon monoxide rapidly impair ...... ng epithelial sodium channels.
@en
P2093
Martin Fronius
Wolfgang G Clauss
Yasmin Buchäckert
P2860
P304
P356
10.1165/RCMB.2008-0458OC
P577
2009-02-27T00:00:00Z