High beta-cell mass prevents streptozotocin-induced diabetes in thioredoxin-interacting protein-deficient mice.
about
New insight into the mechanisms underlying the function of the incretin hormone glucagon-like peptide-1 in pancreatic β-cells: the involvement of the Wnt signaling pathway effector β-cateninThioredoxin/Txnip: redoxisome, as a redox switch for the pathogenesis of diseasesThioredoxin Interacting Protein (TXNIP) and Pathogenesis of Diabetic RetinopathyDisruption of TBP-2 ameliorates insulin sensitivity and secretion without affecting obesity.Thioredoxin binding protein (TBP)-2/Txnip and α-arrestin proteins in cancer and diabetes mellitus.Thioredoxin-Interacting Protein Deficiency Protects against Diabetic Nephropathy.Thioredoxin and thioredoxin target proteins: from molecular mechanisms to functional significance.MiR-17 Downregulation by High Glucose Stabilizes Thioredoxin-Interacting Protein and Removes Thioredoxin Inhibition on ASK1 Leading to Apoptosis.Cytokines Regulate β-Cell Thioredoxin-interacting Protein (TXNIP) via Distinct Mechanisms and PathwaysFOXO1 competes with carbohydrate response element-binding protein (ChREBP) and inhibits thioredoxin-interacting protein (TXNIP) transcription in pancreatic beta cells.Streptozotocin is equally diabetogenic whether administered to fed or fasted miceThioredoxin interacting protein: redox dependent and independent regulatory mechanisms.Toll-like receptors, the NLRP3 inflammasome, and interleukin-1β in the development and progression of type 1 diabetes.Hypomethylation of the promoter of the catalytic subunit of protein phosphatase 2A in response to hyperglycemia.Thioredoxin-interacting protein mediates high glucose-induced reactive oxygen species generation by mitochondria and the NADPH oxidase, Nox4, in mesangial cellsMicroRNAs 106b and 222 Improve Hyperglycemia in a Mouse Model of Insulin-Deficient Diabetes via Pancreatic β-Cell Proliferation.The role of the thioredoxin/thioredoxin reductase system in the metabolic syndrome: towards a possible prognostic marker?W2476 ameliorates β-cell dysfunction and exerts therapeutic effects in mouse models of diabetes via modulation of the thioredoxin-interacting protein signaling pathway.GABA and 5-HT chitosan nanoparticles decrease striatal neuronal degeneration and motor deficits during liver injury.Possible role for the thioredoxin system in the protective effects of probucol in the pancreatic islets of diabetic rats.
P2860
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P2860
High beta-cell mass prevents streptozotocin-induced diabetes in thioredoxin-interacting protein-deficient mice.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
High beta-cell mass prevents s ...... acting protein-deficient mice.
@en
High beta-cell mass prevents s ...... acting protein-deficient mice.
@nl
type
label
High beta-cell mass prevents s ...... acting protein-deficient mice.
@en
High beta-cell mass prevents s ...... acting protein-deficient mice.
@nl
prefLabel
High beta-cell mass prevents s ...... acting protein-deficient mice.
@en
High beta-cell mass prevents s ...... acting protein-deficient mice.
@nl
P2093
P2860
P1476
High beta-cell mass prevents s ...... racting protein-deficient mice
@en
P2093
Edwin P Kwan
Fathima Razik
Herbert Y Gaisano
Howard Goldberg
I George Fantus
Laura Sheu
Shlomit Koren
P2860
P304
P356
10.1152/AJPENDO.90619.2008
P577
2009-02-17T00:00:00Z