Helicobacter pylori cytotoxin-associated gene A activates the signal transducer and activator of transcription 3 pathway in vitro and in vivo. - Wikidata - wikimedia - TriplyDB

Helicobacter pylori cytotoxin-associated gene A activates the signal transducer and activator of transcription 3 pathway in vitro and in vivo.

Helicobacter pylori cytotoxin-associated gene A activates the signal transducer and activator of transcription 3 pathway in vitro and in vivo.

http://www.wikidata.org/entity/Q39894695

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STATs in cancer inflammation and immunity: a leading role for STAT3 Helicobacter pylori infection, oncogenic pathways and epigenetic mechanisms in gastric carcinogenesis.S1PR1-STAT3 signaling is crucial for myeloid cell colonization at future metastatic sites.rs744166 polymorphism of the STAT3 gene is associated with risk of gastric cancer in a Chinese population.IL-22 negatively regulates Helicobacter pylori-induced CCL20 expression in gastric epithelial cells Carcinogenic Helicobacter pylori in gastric pre-cancer and cancer lesions: association with tobacco-chewing Helicobacter pylori CagA phosphorylation status determines the gp130-activated SHP2/ERK and JAK/STAT signal transduction pathways in gastric epithelial cells.Helicobacter pylori CagA triggers expression of the bactericidal lectin REG3γ via gastric STAT3 activation Gene expression profiling in gastric mucosa from Helicobacter pylori-infected and uninfected patients undergoing chronic superficial gastritis Regulation of RKIP function by Helicobacter pylori in gastric cancer.AURKA regulates JAK2-STAT3 activity in human gastric and esophageal cancers.Enhanced IL-6/IL-6R signaling promotes growth and malignant properties in EBV-infected premalignant and cancerous nasopharyngeal epithelial cells.High resolution electron microscopy of the Helicobacter pylori Cag type IV secretion system pili produced in varying conditions of iron availability.Repressed TGF-β signaling through CagA-Smad3 interaction as pathogenic mechanisms of Helicobacter pylori-associated gastritis.Deletion of Stat3 enhances myeloid cell expansion and increases the severity of myeloproliferative neoplasms in Jak2V617F knock-in mice.Cdx2 Expression and Intestinal Metaplasia Induced by H. pylori Infection of Gastric Cells Is Regulated by NOD1-Mediated Innate Immune Responses.The hyaluronic acid receptor CD44 coordinates normal and metaplastic gastric epithelial progenitor cell proliferation Epigenetic silencing of the NR4A3 tumor suppressor, by aberrant JAK/STAT signaling, predicts prognosis in gastric cancer.Helicobacter pylori CagA: From Pathogenic Mechanisms to Its Use as an Anti-Cancer Vaccine.A single nucleotide polymorphism in the 3'-UTR of STAT3 regulates its expression and reduces risk of pancreatic cancer in a Chinese population.Dual roles for immunity in gastrointestinal cancers.Multifaceted link between cancer and inflammation.Targeting STAT3 in gastric cancer.Epigenetic modifications induced by Helicobacter pylori infection through a direct microbe-gastric epithelial cells cross-talk.Influence of H. pylori CagA Coupled with Alcohol Consumption on Cytokine Profiles in Men.Helicobacter pylori virulence factor CagA promotes tumorigenesis of gastric cancer via multiple signaling pathways.STAT3: a critical component in the response to Helicobacter pylori infection.Helicobacter pylori Activates IL-6-STAT3 Signaling in Human Gastric Cancer Cells: Potential Roles for Reactive Oxygen Species.CD24 mediates gastric carcinogenesis and promotes gastric cancer progression via STAT3 activation.A positive feedback loop between STAT3 and cyclooxygenase-2 gene may contribute to Helicobacter pylori-associated human gastric tumorigenesis.STAT3 activation in infection and infection-associated cancer.Type IV Secretion and Signal Transduction of Helicobacter pylori CagA through Interactions with Host Cell Receptors.H. pylori induces the expression of Hath1 in gastric epithelial cells via interleukin-8/STAT3 phosphorylation while suppressing Hes1.Constitutive activation of STAT3 is predictive of poor prognosis in human gastric cancer.α-Lipoic Acid Inhibits Expression of IL-8 by Suppressing Activation of MAPK, Jak/Stat, and NF-κB in H. pylori-Infected Gastric Epithelial AGS Cells.Signature of positive selection of PTK6 gene in East Asian populations: a cross talk for Helicobacter pylori invasion and gastric cancer endemicity.Helicobacter pylori-induced STAT3 activation and signalling network in gastric cancer.Up-regulated Annexin A1 expression in gastrointestinal cancer is associated with cancer invasion and lymph node metastasis.Surreptitious manipulation of the human host by Helicobacter pylori.Streptococcus infantarius and carcinogenesis: a new chapter in colorectal pathology.

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P2860

Helicobacter pylori cytotoxin-associated gene A activates the signal transducer and activator of transcription 3 pathway in vitro and in vivo.

http://www.wikidata.org/entity/Q39894695

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0008-5472.CAN-08-1191

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Cancer Research

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Helicobacter pylori cytotoxin- ...... pathway in vitro and in vivo.

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Aime Franco

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Chihiro Sasakawa

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Dana M Bronte-Tinkew

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Diane Ahn

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Hitomi Mimuro

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Mark J Ropeleski

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Mauricio Terebiznik

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Michelle Ang

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Nicola L Jones

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Richard M Peek

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P2860

Toll-like receptor 3-mediated activation of NF-kappaB and IRF3 diverges at Toll-IL-1 receptor domain-containing adapter inducing IFN-beta

Pathogenesis of Helicobacter pylori infection

Oncogenic mechanisms of the Helicobacter pylori CagA protein

The STATs of cancer--new molecular targets come of age

Stat3 as an oncogene

Helicobacter pylori CagA induces a transition from polarized to invasive phenotypes in MDCK cells.

c-Src/Lyn kinases activate Helicobacter pylori CagA through tyrosine phosphorylation of the EPIYA motifs.

Mucosal FOXP3-expressing CD4+ CD25high regulatory T cells in Helicobacter pylori-infected patients

Activation of beta-catenin by carcinogenic Helicobacter pylori

Mutant cells that do not respond to interleukin-1 (IL-1) reveal a novel role for IL-1 receptor-associated kinase.

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632-639

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10.1158/0008-5472.CAN-08-1191

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2

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19147578

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Helicobacter pylori

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3418672

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