Blockage of angiotensin II type 2 receptor prevents thyroxine-mediated cardiac hypertrophy by blocking Akt activation.
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Role of Oxidative Stress in Thyroid Hormone-Induced Cardiomyocyte Hypertrophy and Associated Cardiac Dysfunction: An Undisclosed StoryEmerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism.Cardiomyocyte-specific deletion of endothelin receptor A rescues aging-associated cardiac hypertrophy and contractile dysfunction: role of autophagyHyperthyroidism and cardiovascular complications: a narrative review on the basis of pathophysiology.Vinexin-β protects against cardiac hypertrophy by blocking the Akt-dependent signalling pathway.MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone.Nardosinone protects H9c2 cardiac cells from angiotensin II-induced hypertrophy.Molecular characterization of thyroid hormone-inhibited atrial L-type calcium channel expression: implication for atrial fibrillation in hyperthyroidism.Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor.Sexual dimorphism in autonomic changes and in the renin-angiotensin system in the hearts of mice subjected to thyroid hormone-induced cardiac hypertrophy.AGTR2 gene polymorphism is associated with muscle fibre composition, athletic status and aerobic performance.Cardioprotective effect of thyroid hormone is mediated by AT2 receptor and involves nitric oxide production via Akt activation in mice.AT1 receptor blockage impairs NF-κB activation mediated by thyroid hormone in cardiomyocytes.Angiotensin II type 2 receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion.Cyclovirobuxinum D alleviates cardiac hypertrophy in hyperthyroid rats by preventing apoptosis of cardiac cells and inhibiting the p38 mitogen-activated protein kinase signaling pathway.Chronic Akt activation accentuates aging-induced cardiac hypertrophy and myocardial contractile dysfunction: role of autophagy.Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy.
P2860
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P2860
Blockage of angiotensin II type 2 receptor prevents thyroxine-mediated cardiac hypertrophy by blocking Akt activation.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
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2010年學術文章
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name
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@en
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@nl
type
label
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@en
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@nl
prefLabel
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@en
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@nl
P2093
P2860
P1476
Blockage of angiotensin II typ ...... hy by blocking Akt activation.
@en
P2093
M L M Barreto-Chaves
M S Carneiro-Ramos
R A S Santos
R L P Vieira
P2860
P2888
P304
P356
10.1007/S00395-010-0089-0
P577
2010-02-14T00:00:00Z