Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha. - Wikidata - wikimedia - TriplyDB

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

http://www.wikidata.org/entity/Q39943758

about

MicroRNAs as Important Players in Host-hepatitis B Virus Interactions Roles of hepatocyte nuclear factors in hepatitis B virus infection Hepatitis B virus molecular biology and pathogenesis MicroRNAs as therapeutic strategy for hepatitis B virus-associated hepatocellular carcinoma: current status and future prospects Prevalent HBV point mutations and mutation combinations at BCP/preC region and their association with liver disease progression.Limited effects of bile acids and small heterodimer partner on hepatitis B virus biosynthesis in vivo.Nuclear receptor variants in liver disease.Synthesis and biological evaluations of chalcones, flavones and chromenes as farnesoid x receptor (FXR) antagonists.Independent activation of hepatitis B virus biosynthesis by retinoids, peroxisome proliferators, and bile acids.Epigenetically regulated miR-449a enhances hepatitis B virus replication by targeting cAMP-responsive element binding protein 5 and modulating hepatocytes phenotype.Multiple nuclear receptors may regulate hepatitis B virus biosynthesis during development.Peroxisome proliferator-activated receptor gamma Coactivator 1alpha and small heterodimer partner differentially regulate nuclear receptor-dependent hepatitis B virus biosynthesis.Transforming growth factor β-activated kinase 1 transcriptionally suppresses hepatitis B virus replication.CRTC2 enhances HBV transcription and replication by inducing PGC1α expression.Hepatocyte metabolic signalling pathways and regulation of hepatitis B virus expression.An unhealthy relationship: viral manipulation of the nuclear receptor superfamily.Interactions of Hepatitis B Virus Infection with Nonalcoholic Fatty Liver Disease: Possible Mechanisms and Clinical Impact.Resveratrol, sirtuins, and viruses.Epigallocatechin gallate inhibits hepatitis B virus via farnesoid X receptor alpha.Virus-host interplay in hepatitis B virus infection and epigenetic treatment strategies.Inhibition of interferon-α-induced signaling by hyperosmolarity and hydrophobic bile acids.Bile Acids Act as Soluble Host Restriction Factors Limiting Cytomegalovirus Replication in Hepatocytes.Bile acids increase hepatitis B virus gene expression and inhibit interferon-alpha activity.Hepatitis B virus: the "metabolovirus" highjacks cholesterol and bile acid metabolism.Bile acid-induced modulation of virus replication.Silencing Retinoid X Receptor Alpha Expression Enhances Early-stage Hepatitis B Virus Infection In Cell Cultures.Anti-inflammatory consequences of bile acid accumulation in virus-infected bile duct ligated mice.Host functions used by hepatitis B virus to complete its life cycle: Implications for developing host-targeting agents to treat chronic hepatitis B

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P2860

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

http://www.wikidata.org/entity/Q39943758

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@en

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@nl

type

label

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@en

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@nl

prefLabel

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@en

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@nl

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P1433

Journal of Virology

P1476

Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.

@en

P2093

Caroline Scholtès

http://www.w3.org/2001/XMLSchema#string

Christophe Ramière

http://www.w3.org/2001/XMLSchema#string

Laure Perrin-Cocon

http://www.w3.org/2001/XMLSchema#string

Mary-Anne Trabaud

http://www.w3.org/2001/XMLSchema#string

Olivier Diaz

http://www.w3.org/2001/XMLSchema#string

Patrice André

http://www.w3.org/2001/XMLSchema#string

Vinca Icard

http://www.w3.org/2001/XMLSchema#string

Vincent Lotteau

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of a nuclear receptor for bile acids

Bile acids: natural ligands for an orphan nuclear receptor

Bile acids for viral hepatitis

Distinct modes of regulation of transcription of hepatitis B virus by the nuclear receptors HNF4alpha and COUP-TF1

Peroxisome proliferator-activated receptor-gamma coactivator 1alpha (PGC-1alpha) regulates triglyceride metabolism by activation of the nuclear receptor FXR

Suppression of hepatitis B virus core promoter by the nuclear orphan receptor TR4

PGC-1alpha: a key regulator of energy metabolism

An efficient one-step site-directed and site-saturation mutagenesis protocol

Hepatitis B virus infection--natural history and clinical consequences

Replication of the wild type and a natural hepatitis B virus nucleocapsid promoter variant is differentially regulated by nuclear hormone receptors in cell culture.

P304

10832-10840

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scholarly article

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10.1128/JVI.00883-08

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21

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82

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2008-09-03T00:00:00Z

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23233803

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18768987

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2573182

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