Dclk1 Defines Quiescent Pancreatic Progenitors that Promote Injury-Induced Regeneration and Tumorigenesis.
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Inflammation Shapes Stem Cells and Stemness during Infection and BeyondThe Origins of Gastric Cancer From Gastric Stem Cells: Lessons From Mouse Models.Mitogen-activated Protein Kinase Kinase Activity Maintains Acinar-to-Ductal Metaplasia and Is Required for Organ Regeneration in Pancreatitis.Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells.A novel antibody against cancer stem cell biomarker, DCLK1-S, is potentially useful for assessing colon cancer risk after screening colonoscopy.PanIN Neuroendocrine Cells Promote Tumorigenesis via Neuronal Cross-talk.Functional implication of Dclk1 and Dclk1-expressing cells in cancer.Acinar cell plasticity and development of pancreatic ductal adenocarcinoma.APSA Awardee Submission: Tumor/cancer stem cell marker doublecortin-like kinase 1 in liver diseases.Targeting cancer stem cells for chemoprevention of pancreatic cancer.Cells of origin of pancreatic neoplasms.Epigenetic regulation of human DCLK-1 gene during colon-carcinogenesis: clinical and mechanistic implicationsDoublecortin and CaM kinase-like-1 as an independent prognostic factor in patients with resected pancreatic carcinoma.Epithelial Dclk1+ cells are not neural crest derived.Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors.Identification of quiescent and spatially restricted mammary stem cells that are hormone responsive.Repurposing Established Compounds to Target Pancreatic Cancer Stem Cells (CSCs).Identification of Newly Committed Pancreatic Cells in the Adult Mouse Pancreas.Cell plasticity in epithelial homeostasis and tumorigenesis.Expression of doublecortin and CaM kinase-like-1 protein in serrated neoplasia of the colorectum.Pancreatic β Cell Regeneration as a Possible Therapy for Diabetes.DCAMKL1 is associated with the malignant status and poor outcome in bladder cancer.Immune Cell Production of Interleukin 17 Induces Stem Cell Features of Pancreatic Intraepithelial Neoplasia Cells.Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice.Heterogeneity of SOX9 and HNF1β in Pancreatic Ducts Is Dynamic.Pancreatic Cancer: Molecular Characterization, Clonal Evolution and Cancer Stem Cells.Tumorigenic Cell Reprogramming and Cancer Plasticity: Interplay between Signaling, Microenvironment, and Epigenetics.Expansion of transplanted islets in mice by co-transplantation with adipose tissue-derived mesenchymal stem cells.Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment.Doublecortin-like kinase 1 compromises DNA repair and induces chromosomal instability
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P2860
Dclk1 Defines Quiescent Pancreatic Progenitors that Promote Injury-Induced Regeneration and Tumorigenesis.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh
2016年學術文章
@zh-hant
name
Dclk1 Defines Quiescent Pancre ...... egeneration and Tumorigenesis.
@en
type
label
Dclk1 Defines Quiescent Pancre ...... egeneration and Tumorigenesis.
@en
prefLabel
Dclk1 Defines Quiescent Pancre ...... egeneration and Tumorigenesis.
@en
P2093
P2860
P50
P1433
P1476
Dclk1 Defines Quiescent Pancre ...... egeneration and Tumorigenesis.
@en
P2093
Aleksandra M Urbanska
Anil K Rustgi
Barry Honig
Bernhard W Renz
C Benedikt Westphalen
Courtney W Houchen
Daniel L Worthley
Gloria H Su
Joshua Broyde
Karan Nagar
P2860
P304
P356
10.1016/J.STEM.2016.03.016
P407
P577
2016-04-01T00:00:00Z