Co-overexpression of Janus kinase 2 and signal transducer and activator of transcription 5a promotes differentiation of mammary cancer cells through reversal of epithelial-mesenchymal transition.
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Low levels of Stat5a protein in breast cancer are associated with tumor progression and unfavorable clinical outcomesConditional knockout of fibronectin abrogates mouse mammary gland lobuloalveolar differentiation.Prolactin inhibits BCL6 expression in breast cancer through a Stat5a-dependent mechanism.Role of ERalpha in the differential response of Stat5a loss in susceptibility to mammary preneoplasia and DMBA-induced carcinogenesisExpression of prolactin receptors in normal canine mammary tissue, canine mammary adenomas and mammary adenocarcinomas.PTP1B suppresses prolactin activation of Stat5 in breast cancer cells.Signal transducer and activator of transcription-3 and breast cancer prognosis.Three interrelated themes in current breast cancer research: gene addiction, phenotypic plasticity, and cancer stem cells.Signal transducer and activator of transcription 5 as a key signaling pathway in normal mammary gland developmental biology and breast cancer.Endocrine control of canine mammary neoplasms: serum reproductive hormone levels and tissue expression of steroid hormone, prolactin and growth hormone receptors.Steroid induction of therapy-resistant cytokeratin-5-positive cells in estrogen receptor-positive breast cancer through a BCL6-dependent mechanism.Truncated p110 ERBB2 induces mammary epithelial cell migration, invasion and orthotopic xenograft formation, and is associated with loss of phosphorylated STAT5Loss of nuclear localized and tyrosine phosphorylated Stat5 in breast cancer predicts poor clinical outcome and increased risk of antiestrogen therapy failure.Global profiling of prolactin-modulated transcripts in breast cancer in vivo.Prolactin suppresses a progestin-induced CK5-positive cell population in luminal breast cancer through inhibition of progestin-driven BCL6 expression.Prolactin-Stat5 signaling in breast cancer is potently disrupted by acidosis within the tumor microenvironment.Nuclear JAK2: form and function in cancer.Stat5 in breast cancer: potential oncogenic activity coincides with positive prognosis for the disease.Trigonella foenum (Fenugreek) Induced Apoptosis in Hepatocellular Carcinoma Cell Line, HepG2, Mediated by Upregulation of p53 and Proliferating Cell Nuclear Antigen.Phosphorylation of calcium/calmodulin-stimulated protein kinase II at T286 enhances invasion and migration of human breast cancer cells.Decrease of lactogenic hormones induce epithelial-mesenchymal transition via TGFβ1 and arachidonic acid during mammary gland involution.Stromal-epithelial interactions modulate cross-talk between prolactin receptor and HER2/Neu in breast cancer.EPHB6 augments both development and drug sensitivity of triple-negative breast cancer tumours.
P2860
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P2860
Co-overexpression of Janus kinase 2 and signal transducer and activator of transcription 5a promotes differentiation of mammary cancer cells through reversal of epithelial-mesenchymal transition.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Co-overexpression of Janus kin ...... helial-mesenchymal transition.
@en
type
label
Co-overexpression of Janus kin ...... helial-mesenchymal transition.
@en
prefLabel
Co-overexpression of Janus kin ...... helial-mesenchymal transition.
@en
P2093
P2860
P1433
P1476
Co-overexpression of Janus kin ...... helial-mesenchymal transition.
@en
P2093
Ahmed S Sultan
Hassan Brim
Zaki A Sherif
P2860
P304
P356
10.1111/J.1349-7006.2007.00685.X
P577
2008-02-01T00:00:00Z