Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells. - Wikidata - wikimedia - TriplyDB

Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.

Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.

http://www.wikidata.org/entity/Q40020900

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Interactive effect of cigarette smoke extract and World Trade Center dust particles on airway cell cytotoxicity Environmental risk factors for multiple sclerosis: a review with a focus on molecular mechanisms Respiratory epithelial cells orchestrate pulmonary innate immunity.Impact of perinatal environmental tobacco smoke on the development of childhood allergic diseases Indoor pollutant exposures modify the effect of airborne endotoxin on asthma in urban children Protocol for a human in vivo model of acute cigarette smoke inhalation challenge in smokers with COPD: monitoring the nasal and systemic immune response using a network biology approach Beta defensin-2 is reduced in central but not in distal airways of smoker COPD patients Resveratrol reduces the apoptosis induced by cigarette smoke extract by upregulating MFN2.PPARγ agonists regulate tobacco smoke-induced Toll like receptor 4 expression in alveolar macrophages.Maturation of innate responses to mycobacteria over the first nine months of life.Mechanistic Evaluation of the Impact of Smoking and Chronic Obstructive Pulmonary Disease on the Nasal Epithelium Tobacco smoke mediated induction of sinonasal microbial biofilms Molecular processes that drive cigarette smoke-induced epithelial cell fate of the lung.Cigarette smoke and lipopolysaccharide induce a proliferative airway smooth muscle phenotype Bone marrow mononuclear cells up-regulate toll-like receptor expression and produce inflammatory mediators in response to cigarette smoke extract Regulation of Human MUC7 Mucin Gene Expression by Cigarette Smoke Extract or Cigarette Smoke and Pseudomonas aeruginosa Lipopolysaccharide in Human Airway Epithelial Cells and in MUC7 Transgenic Mice.Comparison of nasal and bronchial epithelial cells obtained from patients with COPD Tobacco smoke exposure and altered nasal responses to live attenuated influenza virus.Whole cigarette smoke increased the expression of TLRs, HBDs, and proinflammory cytokines by human gingival epithelial cells through different signaling pathways.Inhibition by cigarette smoke of nuclear factor-κB-dependent response to bacteria in the airway.Effects in cigarette smoke stimulated bronchial epithelial cells of a corticosteroid entrapped into nanostructured lipid carriers.Effects of air pollutants on innate immunity: the role of Toll-like receptors and nucleotide-binding oligomerization domain-like receptors.Smoking as a permissive factor of periodontal disease in psoriasis.Cigarette smoke condensate modulates migration of human gingival epithelial cells and their interactions with Porphyromonas gingivalis.Genetic ablation of the aryl hydrocarbon receptor causes cigarette smoke-induced mitochondrial dysfunction and apoptosis Cigarette smoke alters the proteomic profile of lung fibroblasts.Cigarette smoke increases TLR4 and TLR9 expression and induces cytokine production from CD8(+) T cells in chronic obstructive pulmonary disease.Cigarette smoking and inflammation: cellular and molecular mechanisms.Alteration of the nasal responses to influenza virus by tobacco smoke Blockade of Interplay between IL-17A and Endoplasmic Reticulum Stress Attenuates LPS-Induced Lung Injury Effect of Tobacco Smoking on The Clinical, Histopathological, and Serological Manifestations of Sjögren's Syndrome.The risk of smoking on multiple sclerosis: a meta-analysis based on 20,626 cases from case-control and cohort studies Comparative cytoprotective effects of carbocysteine and fluticasone propionate in cigarette smoke extract-stimulated bronchial epithelial cells.Characterization of TLR-induced inflammatory responses in COPD and control lung tissue explants.Cigarette smoke enhances human rhinovirus-induced CXCL8 production via HuR-mediated mRNA stabilization in human airway epithelial cells.Advances in Gut Microbiome Research, Opening New Strategies to Cope with a Western Lifestyle.Innate immunity in the respiratory epithelium.The airway epithelium in asthma.The interaction between mother and fetus and the development of allergic asthma.Immunobiology of critical pediatric asthma.

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P2860

Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.

http://www.wikidata.org/entity/Q40020900

description

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Cigarette smoke increases Toll ...... ses in airway epithelial cells

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Angela M Montalbano

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Elisabetta Pace

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Giovanni Bonsignore

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Malcolm Johnson

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Maria Ferraro

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Maria R Bonsignore

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Mark Gjomarkaj

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P2860

Effects of cigarette smoke condensate on proliferation and wound closure of bronchial epithelial cells in vitro: role of glutathione

Toll-like receptors in the induction of the innate immune response

Toll receptors in innate immunity

Inhibition of interleukin 1 receptor/Toll-like receptor signaling through the alternatively spliced, short form of MyD88 is due to its failure to recruit IRAK-4

Bacterial infection in chronic obstructive pulmonary disease in 2000: a state-of-the-art review

Evidence of bacterial infection in acute exacerbations of chronic bronchitis.

The bronchial epithelium in asthma--much more than a passive barrier.

Activation of airway epithelial cells by toll-like receptor agonists.

Bacteria, antibiotics and COPD.

Molecular mechanisms of NF-kappaB activation induced by bacterial lipopolysaccharide through Toll-like receptors.

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