Uncoupling the coupled calcium and zinc dyshomeostasis in cardiac myocytes and mitochondria seen in aldosteronism. - Wikidata - wikimedia - TriplyDB

Uncoupling the coupled calcium and zinc dyshomeostasis in cardiac myocytes and mitochondria seen in aldosteronism.

Uncoupling the coupled calcium and zinc dyshomeostasis in cardiac myocytes and mitochondria seen in aldosteronism.

http://www.wikidata.org/entity/Q40026105

about

Zinc and the prooxidant heart failure phenotype Cellular and molecular pathways to myocardial necrosis and replacement fibrosis.Calcium and zinc dyshomeostasis during isoproterenol-induced acute stressor state.Cation dyshomeostasis and cardiomyocyte necrosis: the Fleckenstein hypothesis revisited Intracellular calcium overloading and oxidative stress in cardiomyocyte necrosis via a mitochondriocentric signal-transducer-effector pathway.Disturbances in calcium metabolism and cardiomyocyte necrosis: the role of calcitropic hormones.Mineralocorticoid receptor antagonism confers cardioprotection in heart failure.Gene Expression Profiles of Peripheral Blood Mononuclear Cells Reveal Transcriptional Signatures as Novel Biomarkers for Cardiac Remodeling in Rats with Aldosteronism and Hypertensive Heart Disease.Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes.Fibrosis in hypertensive heart disease: molecular pathways and cardioprotective strategies.From aldosteronism to oxidative stress: the role of excessive intracellular calcium accumulation.Stressor states and the cation crossroads.Mitochondria play a central role in nonischemic cardiomyocyte necrosis: common to acute and chronic stressor states.Aldosterone and parathyroid hormone: a precarious couple for cardiovascular disease.Heart failure as a risk factor for osteoporosis and fractures.Atrophied cardiomyocytes and their potential for rescue and recovery of ventricular function.Atrophic cardiomyocyte signaling in hypertensive heart disease Small dedifferentiated cardiomyocytes bordering on microdomains of fibrosis: evidence for reverse remodeling with assisted recovery.Mitochondria-targeted cardioprotection in aldosteronism.Congestive heart failure: where homeostasis begets dyshomeostasis.Mitochondriocentric pathway to cardiomyocyte necrosis in aldosteronism: cardioprotective responses to carvedilol and nebivolol.Impact of Labile Zinc on Heart Function: From Physiology to Pathophysiology.Influence of renal impairment on aldosterone status, calcium metabolism, and vasopressin activity in outpatients with systolic heart failure.

P2860

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P2860

Uncoupling the coupled calcium and zinc dyshomeostasis in cardiac myocytes and mitochondria seen in aldosteronism.

http://www.wikidata.org/entity/Q40026105

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

Uncoupling the coupled calcium ...... hondria seen in aldosteronism.

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type

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Uncoupling the coupled calcium ...... hondria seen in aldosteronism.

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Uncoupling the coupled calcium ...... hondria seen in aldosteronism.

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FJC.0B013E3181CF0090

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Journal of Cardiovascular Pharmacology

P1476

Uncoupling the coupled calcium ...... hondria seen in aldosteronism.

@en

P2093

Atta U Shahbaz

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German Kamalov

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Ivan C Gerling

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Karl T Weber

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Patti L Johnson

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Robert A Ahokas

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Syamal K Bhattacharya

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Tieqiang Zhao

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Wenyuan Zhao

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Yao Sun

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P2860

Antioxidant micronutrients in major trauma and burns: evidence and practice

Isoproterenol-induced myocardial fibrosis in relation to myocyte necrosis

Zinc accumulation in adriamycin-induced cardiomyopathy in rats: effects of melatonin, a cardioprotective antioxidant

Regulation of metallothionein gene expression by oxidative stress and metal ions

Rationale and design of the pediatric critical illness stress-induced immune suppression (CRISIS) prevention trial.

Temporal responses to intrinsically coupled calcium and zinc dyshomeostasis in cardiac myocytes and mitochondria during aldosteronism.

The function of zinc metallothionein: a link between cellular zinc and redox state.

Structural basis of end-stage failure in ischemic cardiomyopathy in humans.

The general adaptation syndrome and the diseases of adaptation.

Reduction of nosocomial pneumonia after major burns by trace element supplementation: aggregation of two randomised trials.

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248-254

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20051880

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