Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
about
Spreading the word: non-autonomous effects of apoptosis during development, regeneration and diseaseNew roles for old enzymes: killer caspases as the engine of cell behavior changesKilling me softly--future challenges in apoptosis researchThe core apoptotic executioner proteins CED-3 and CED-4 promote initiation of neuronal regeneration in Caenorhabditis elegansPutting the pieces together: How is the mitochondrial pathway of apoptosis regulated in cancer and chemotherapy?M867, a novel selective inhibitor of caspase-3 enhances cell death and extends tumor growth delay in irradiated lung cancer modelsA nanoconjugate Apaf-1 inhibitor protects mesothelial cells from cytokine-induced injuryPaclitaxel promotes a caspase 8-mediated apoptosis through death effector domain association with microtubules.Radiosensitization of solid tumors by Z-VAD, a pan-caspase inhibitor.Bax regulates production of superoxide in both apoptotic and nonapoptotic neurons: role of caspasesRemoval of Metabolic Liabilities Enables Development of Derivatives of Procaspase-Activating Compound 1 (PAC-1) with Improved PharmacokineticsDerivatives of Procaspase-Activating Compound 1 (PAC-1) and their Anticancer Activities.The HMGB1 protein induces a metabolic type of tumour cell death by blocking aerobic respiration.Activation of executioner caspases is a predictor of progression-free survival in glioblastoma patients: a systems medicine approach.c-Met expression and activity in urogenital cancers - novel aspects of signal transduction and medical implications.Caspase-8 as a therapeutic target in cancer.New frontiers in promoting tumour cell death: targeting apoptosis, necroptosis and autophagy.Cell death-stimulated cell proliferation: a tissue regeneration mechanism usurped by tumors during radiotherapyThe effect of silver nanoparticles (AgNPs) on proliferation and apoptosis of in ovo cultured glioblastoma multiforme (GBM) cells.Caspase-3 expression in tumorigenesis and prognosis of buccal mucosa squamous cell carcinoma.Synergistic and targeted therapy with a procaspase-3 activator and temozolomide extends survival in glioma rodent models and is feasible for the treatment of canine malignant glioma patients.Inhibition of TDP43-Mediated SNHG12-miR-195-SOX5 Feedback Loop Impeded Malignant Biological Behaviors of Glioma Cells.MRSI-based molecular imaging of therapy response to temozolomide in preclinical glioblastoma using source analysis.Learning on the Fly: The Interplay between Caspases and Cancer.Caspase-3 and Bcl-2 expression in glioblastoma: an immunohistochemical study.
P2860
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P2860
Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
@en
type
label
Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
@en
prefLabel
Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
@en
P2093
P1476
Basal caspase activity promotes migration and invasiveness in glioblastoma cells.
@en
P2093
Anika Eckert
Barbara C Böck
Benedict Wiestler
Benjamin Funke
Georg Gdynia
Kerstin Grund
Otmar D Wiestler
Sebastian Sieber
Stephan Macher-Goeppinger
Wilfried Roth
P304
P356
10.1158/1541-7786.MCR-07-0343
P577
2007-12-01T00:00:00Z