Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
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Expression of DDX3 is directly modulated by hypoxia inducible factor-1 alpha in breast epithelial cellsHyaluronan-CD44 interaction promotes c-Src-mediated twist signaling, microRNA-10b expression, and RhoA/RhoC up-regulation, leading to Rho-kinase-associated cytoskeleton activation and breast tumor cell invasionEpithelial-mesenchymal transition in cancer: parallels between normal development and tumor progressionTwist modulates breast cancer stem cells by transcriptional regulation of CD24 expressionHijacking the Hexosamine Biosynthetic Pathway to Promote EMT-Mediated Neoplastic PhenotypesNovel Molecular Markers for Breast CancerClaudin 1 in Breast Cancer: New InsightsNormal and disease-related biological functions of Twist1 and underlying molecular mechanismsBreast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticityRegulation of epithelial-mesenchymal transition through epigenetic and post-translational modificationsAKT-ions with a TWIST between EMT and METFunctional Analysis of Two Novel Mutations in TWIST1 Protein Motifs Found in Ventricular Septal Defect PatientsTwist: a molecular target in cancer therapeuticsTwist1-induced dissemination preserves epithelial identity and requires E-cadherin.Regulation of the Epithelial-Mesenchymal Transition by Claudin-3 and Claudin-4miR-151-3p Targets TWIST1 to Repress Migration of Human Breast Cancer CellsRestoration of E-cadherin cell-cell junctions requires both expression of E-cadherin and suppression of ERK MAP kinase activation in Ras-transformed breast epithelial cells.Sub-circuits of a gene regulatory network control a developmental epithelial-mesenchymal transitionDACH1 inhibits SNAI1-mediated epithelial-mesenchymal transition and represses breast carcinoma metastasis.Monoamine oxidase A mediates prostate tumorigenesis and cancer metastasis.Regulation of cell proliferation and migration by p62 through stabilization of Twist1.EMT and MET: necessary or permissive for metastasis?Cellular migration and invasion uncoupled: increased migration is not an inexorable consequence of epithelial-to-mesenchymal transitionCoordinated epigenetic repression of the miR-200 family and miR-205 in invasive bladder cancer.TWIST1 a new determinant of epithelial to mesenchymal transition in EGFR mutated lung adenocarcinomaIntratumoral macrophages contribute to epithelial-mesenchymal transition in solid tumors.TWIST1 associates with NF-κB subunit RELA via carboxyl-terminal WR domain to promote cell autonomous invasion through IL8 productionCyr61 promotes epithelial-mesenchymal transition and tumor metastasis of osteosarcoma by Raf-1/MEK/ERK/Elk-1/TWIST-1 signaling pathway.Slug, twist, and E-cadherin as immunohistochemical biomarkers in meningeal tumors.Transcriptional repression of E-cadherin by human papillomavirus type 16 E6.Anchoring junctions as drug targets: role in contraceptive development.Epithelial-mesenchymal transition stimulates human cancer cells to extend microtubule-based invasive protrusions and suppresses cell growth in collagen gel.Sox2 suppresses the invasiveness of breast cancer cells via a mechanism that is dependent on Twist1 and the status of Sox2 transcription activity.Abnormal expression of serum soluble E-cadherin is correlated with clinicopathological features and prognosis of breast cancer.Critical roles of p53 in epithelial-mesenchymal transition and metastasis of hepatocellular carcinoma cells.Phosphorylation of serine 68 of Twist1 by MAPKs stabilizes Twist1 protein and promotes breast cancer cell invasivenessImpact of p120-catenin isoforms 1A and 3A on epithelial mesenchymal transition of lung cancer cells expressing E-cadherin in different subcellular locationsRNA-based TWIST1 inhibition via dendrimer complex to reduce breast cancer cell metastasis.Cadherin-catenin complex dissociation in lobular neoplasia of the breastCx31.1 acts as a tumour suppressor in non-small cell lung cancer (NSCLC) cell lines through inhibition of cell proliferation and metastasis
P2860
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P2860
Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
@en
type
label
Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
@en
prefLabel
Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
@en
P2093
P2860
P1476
Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer.
@en
P2093
Ji Hshiung Chen
Paul van Diest
Venu Raman
P2860
P304
P356
10.1016/J.BBRC.2007.11.151
P577
2007-12-04T00:00:00Z