about
TLRs control hematopoiesis during infectionCandida albicans induces selective development of macrophages and monocyte derived dendritic cells by a TLR2 dependent signallingGut microbiota promote hematopoiesis to control bacterial infectionInterferon regulatory factor 8 and the regulation of neutrophil, monocyte, and dendritic cell production.Role of Toll-like receptors in systemic Candida albicans infections.Characterization of a new murine retinal cell line (MU-PH1) with glial, progenitor and photoreceptor characteristics.TLR2, TLR4 and Dectin-1 signalling in hematopoietic stem and progenitor cells determines the antifungal phenotype of the macrophages they produce.Autocrine Type I IFN Signaling in Dendritic Cells Stimulated with Fungal β-Glucans or Lipopolysaccharide Promotes CD8 T Cell Activation.C9orf72 is required for proper macrophage and microglial function in mice.Detection of a TLR2 agonist by hematopoietic stem and progenitor cells impacts the function of the macrophages they produce.IRF8 acts in lineage-committed rather than oligopotent progenitors to control neutrophil vs monocyte production.Dectin-1 mediates in vitro phagocytosis of Candida albicans yeast cells by retinal microglia.Counting the cost of lineage decisions.Granulocyte-Monocyte Progenitors and Monocyte-Dendritic Cell Progenitors Independently Produce Functionally Distinct Monocytes.PRR signaling during in vitro macrophage differentiation from progenitors modulates their subsequent response to inflammatory stimuli.Candida albicans triggers proliferation and differentiation of hematopoietic stem and progenitor cells by a MyD88-dependent signaling.Systemic Candidiasis and TLR2 Agonist Exposure Impact the Antifungal Response of Hematopoietic Stem and Progenitor CellsBoth viable and killed Candida albicans cells induce in vitro production of TNF-alpha and IFN-gamma in murine cells through a TLR2-dependent signallingIn vitro response to Candida albicans in cultures of whole human blood from young and aged donorsEnhanced proinflammatory response to the Candida albicans gpi7 null mutant by murine cellsInfluence of aging on murine neutrophil and macrophage function against Candida albicansMyD88 is dispensable for resistance to Paracoccidioides brasiliensis in a murine model of blood-borne disseminated infectionSignalling through TLR2/MyD88 induces differentiation of murine bone marrow stem and progenitor cells to functional phagocytes in response to Candida albicansIdentification and Isolation of Oligopotent and Lineage-committed Myeloid Progenitors from Mouse Bone MarrowTLR2 and Dectin-1 Signaling in Mouse Hematopoietic Stem and Progenitor Cells Impacts the Ability of the Antigen Presenting Cells They Produce to Activate CD4 T Cells
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Alberto Yáñez
@ast
Alberto Yáñez
@en
Alberto Yáñez
@es
Alberto Yáñez
@nl
type
label
Alberto Yáñez
@ast
Alberto Yáñez
@en
Alberto Yáñez
@es
Alberto Yáñez
@nl
prefLabel
Alberto Yáñez
@ast
Alberto Yáñez
@en
Alberto Yáñez
@es
Alberto Yáñez
@nl
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P21
P31
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0000-0002-7719-1963