Whole Wiskott‑Aldrich syndrome protein gene deletion identified by high throughput sequencing.

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Whole Wiskott‑Aldrich syndrome protein gene deletion identified by high throughput sequencing.

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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Whole Wiskott‑Aldrich syndrome ...... by high throughput sequencing.

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label

Whole Wiskott‑Aldrich syndrome ...... by high throughput sequencing.

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prefLabel

Whole Wiskott‑Aldrich syndrome ...... by high throughput sequencing.

@en

P1476

Whole Wiskott‑Aldrich syndrome ...... by high throughput sequencing.

@en

P2093

Bing Zhang

http://www.w3.org/2001/XMLSchema#string

Runying Zou

http://www.w3.org/2001/XMLSchema#string

Xiangling He

http://www.w3.org/2001/XMLSchema#string

Xin Tian

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Yalan You

http://www.w3.org/2001/XMLSchema#string

Yang Yang

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P2860

A hydrophobic pocket in the active site of glycolytic aldolase mediates interactions with Wiskott-Aldrich syndrome protein

Isolation of a novel gene mutated in Wiskott-Aldrich syndrome

The human Arp2/3 complex is composed of evolutionarily conserved subunits and is localized to cellular regions of dynamic actin filament assembly

Fast and accurate short read alignment with Burrows-Wheeler transform

Cortactin localization to sites of actin assembly in lamellipodia requires interactions with F-actin and the Arp2/3 complex.

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Structural and biochemical characterization of two binding sites for nucleation-promoting factor WASp-VCA on Arp2/3 complex

X-linked thrombocytopenia caused by a mutation in the Wiskott-Aldrich syndrome (WAS) gene that disrupts interaction with the WAS protein (WASP)-interacting protein (WIP)

Osmotic stress-induced remodeling of the cortical cytoskeleton

A novel primary human immunodeficiency due to deficiency in the WASP-interacting protein WIP

P304

6526-6531

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P31

scholarly article

P356

10.3892/MMR.2017.7416

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P577

2017-08-31T00:00:00Z

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P698

28901403

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P932

5865821

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