Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

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Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

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Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

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Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

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P1476

Bcl10 synergistically links CEACAM3 and TLR-dependent inflammatory signalling.

@en

P2093

Anna Sintsova

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Cynthia X Guo

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Helen Sarantis

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Michael Glogauer

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Tak W Mak

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P2860

Bcl10 is a positive regulator of antigen receptor-induced activation of NF-kappaB and neural tube closure

The IRAK-1-BCL10-MALT1-TRAF6-TAK1 cascade mediates signaling to NF-kappaB from Toll-like receptor 4

Functional and receptor binding characterization of recombinant murine macrophage inflammatory protein 2: sequence analysis and mutagenesis identify receptor binding epitopes.

Granulocyte CEACAM3 is a phagocytic receptor of the innate immune system that mediates recognition and elimination of human-specific pathogens

Card9 controls a non-TLR signalling pathway for innate anti-fungal immunity

Differential requirement for Malt1 in T and B cell antigen receptor signaling

Global analysis of neutrophil responses to Neisseria gonorrhoeae reveals a self-propagating inflammatory program

Reprint of Neutrophil cell surface receptors and their intracellular signal transduction pathways.

Opa+ Neisseria gonorrhoeae exhibits reduced survival in human neutrophils via Src family kinase-mediated bacterial trafficking into mature phagolysosomes

Assembly of NADPH oxidase in human neutrophils is modulated by the opacity-associated protein expression State of Neisseria gonorrhoeae

P31

scholarly article

P356

10.1111/CMI.12788

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P50

Scott D Gray-Owen

P577

2017-09-08T00:00:00Z

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P698

28886618

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P921

Toll-like receptor