Downregulation of CCN3 expression as a potential mechanism for melanoma progression. - Wikidata - wikimedia - TriplyDB

Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

http://www.wikidata.org/entity/Q40060499

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CCN3 suppresses mitogenic signalling and reinstates growth control mechanisms in Chronic Myeloid Leukaemia CCN proteins: A centralized communication network CCN3 impairs osteoblast and stimulates osteoclast differentiation to favor breast cancer metastasis to bone The CCN family of proteins: structure-function relationships Crosstalk in skin: melanocytes, keratinocytes, stem cells, and melanoma NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9 Matricellular protein CCN3 (NOV) regulates actin cytoskeleton reorganization.Cancer biomarker discovery: the entropic hallmark.Reduced NOV expression correlates with disease progression in colorectal cancer and is associated with survival, invasion and chemoresistance of cancer cells hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3.Inhibition of fibroblast growth by Notch1 signaling is mediated by induction of Wnt11-dependent WISP-1.Plasma NOV/CCN3 levels are closely associated with obesity in patients with metabolic disorders.Huaier restrains proliferative and invasive potential of human hepatoma SKHEP-1 cells partially through decreased Lamin B1 and elevated NOV.CCN3 and bone marrow cells.Mechanisms for reaching the differentiated state: Insights from neural crest-derived melanocytes.Differential expression of CCN family members CYR611, CTGF and NOV in gastric cancer and their association with disease progression The novel secreted factor MIG-18 acts with MIG-17/ADAMTS to control cell migration in Caenorhabditis elegans.Periostin is required for matricellular localization of CCN3 in periodontal ligament of mice.Microenvironment and tumor progression of melanoma: new therapeutic prospectives.CCN3/NOV gene expression in human prostate cancer is directly suppressed by the androgen receptor.Emerging role of CCN family proteins in tumorigenesis and cancer metastasis (Review).The epidermal polarity protein Par3 is a non-cell autonomous suppressor of malignant melanoma.Modulation of the pro-inflammatory cytokines and matrix metalloproteinases production in co-cultivated human keratinocytes and melanocytes.CCN3 promotes prostate cancer bone metastasis by modulating the tumor-bone microenvironment through RANKL-dependent pathway.CCN3 (NOV) regulates proliferation, adhesion, migration and invasion in clear cell renal cell carcinoma CCN2 expression and localization in melanoma cells.CCN3: a key growth regulator in Chronic Myeloid Leukaemia.Regulatory role of CCN3 in melanoma cell interaction with the extracellular matrix.CCN3: the-pain-killer inside me.Is primary melanoma ulceration a factor of good response to adoptive immunotherapy?CCN3-mediated promotion of sulfated proteoglycan synthesis in rat chondrocytes from developing joint heads.The matricellular protein CCN3 regulates NOTCH1 signalling in chronic myeloid leukaemia.Matricellular proteins produced by melanocytes and melanomas: in search for functions.CCN3 promotes epithelial-mesenchymal transition in prostate cancer via FAK/Akt/HIF-1α-induced twist expression.CCN3 suppresses TGF-β1-induced extracellular matrix accumulation in human mesangial cells in vitro.

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Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

http://www.wikidata.org/entity/Q40060499

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2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

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Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

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Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

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Downregulation of CCN3 expression as a potential mechanism for melanoma progression.

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B Perbal

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D E Elder

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G Martinez

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I Juhasz

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K Balint

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M Fukunaga-Kalabis

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P2860

Inhibition of glioma cell growth and tumorigenic potential by CCN3 (NOV)

Thickness, cross-sectional areas and depth of invasion in the prognosis of cutaneous melanoma

Tumour progression and the nature of cancer

CCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motility

A novel mechanism for BCR-ABL action: stimulated secretion of CCN3 is involved in growth and differentiation regulation

In Ewing's sarcoma CCN3(NOV) inhibits proliferation while promoting migration and invasion of the same cell type

NOV (nephroblastoma overexpressed) and the CCN family of genes: structural and functional issues.

Molecular role(s) for integrins in human melanoma invasion.

Differential expression of the ccn3 (nov) proto-oncogene in human prostate cell lines and tissues.

Adhesion, migration and communication in melanocytes and melanoma.

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10.1038/SJ.ONC.1210896

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18

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27

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1002435276

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17968313

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