Luteolin, quercetin, genistein and quercetagetin inhibit the effects of lipopolysaccharide obtained from Porphyromonas gingivalis in H9c2 cardiomyoblasts.

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Luteolin, quercetin, genistein and quercetagetin inhibit the effects of lipopolysaccharide obtained from Porphyromonas gingivalis in H9c2 cardiomyoblasts.

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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name

Luteolin, quercetin, genistein ...... valis in H9c2 cardiomyoblasts.

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type

Item

label

Luteolin, quercetin, genistein ...... valis in H9c2 cardiomyoblasts.

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prefLabel

Luteolin, quercetin, genistein ...... valis in H9c2 cardiomyoblasts.

@en

P1476

Luteolin, quercetin, genistein ...... valis in H9c2 cardiomyoblasts.

@en

P2093

Alfredo Torras-Ceballos

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Gloria Gutiérrez-Venegas

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Juan Arturo Gómez-Mora

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P2860

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

Luteolin inhibits an endotoxin-stimulated phosphorylation cascade and proinflammatory cytokine production in macrophages.

Inhibitory effects of flavonoids on phosphodiesterase isozymes from guinea pig and their structure-activity relationships.

MAPK signaling drives inflammation in LPS-stimulated cardiomyocytes: the route of crosstalk to G-protein-coupled receptors.

Poor oral hygiene as a risk factor for infective endocarditis-related bacteremia.

Pachymic acid protects H9c2 cardiomyocytes from lipopolysaccharide-induced inflammation and apoptosis by inhibiting the extracellular signal-regulated kinase 1/2 and p38 pathways.

Anti-inflammatory effects of genistein via suppression of the toll-like receptor 4-mediated signaling pathway in lipopolysaccharide-stimulated BV2 microglia.

Quercetin disrupts tyrosine-phosphorylated phosphatidylinositol 3-kinase and myeloid differentiation factor-88 association, and inhibits MAPK/AP-1 and IKK/NF-κB-induced inflammatory mediators production in RAW 264.7 cells.

Molecular mechanism of capillarisin-mediated inhibition of MyD88/TIRAP inflammatory signaling in in vitro and in vivo experimental models.

P2888

s11658-017-0047-z

P304

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P31

scholarly article

P356

10.1186/S11658-017-0047-Z

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P478

http://www.w3.org/2001/XMLSchema#string

P50

Berenice Fernández-Rojas

P577

2017-09-04T00:00:00Z

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P6179

1091481756

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P698

28878808

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P921

Porphyromonas gingivalis

lipopolysaccharide

P932

5583969

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