N-3 PUFAs induce inflammatory tolerance by formation of KEAP1-containing SQSTM1/p62-bodies and activation of NFE2L2.

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N-3 PUFAs induce inflammatory tolerance by formation of KEAP1-containing SQSTM1/p62-bodies and activation of NFE2L2.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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N-3 PUFAs induce inflammatory ...... dies and activation of NFE2L2.

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N-3 PUFAs induce inflammatory ...... dies and activation of NFE2L2.

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N-3 PUFAs induce inflammatory ...... dies and activation of NFE2L2.

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P1476

N-3 PUFAs induce inflammatory ...... dies and activation of NFE2L2.

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P2093

Babak Razani

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Eli Kjøbli

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Ida Johansson

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Ismail Sergin

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Jennifer Mildenberger

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Trude Helen Flo

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P2860

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction

Phosphorylation of the autophagy receptor optineurin restricts Salmonella growth

p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

Essential role for TAX1BP1 in the termination of TNF-alpha-, IL-1- and LPS-mediated NF-kappaB and JNK signaling

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

Identification of a novel macrophage phenotype that develops in response to atherogenic phospholipids via Nrf2.

GPR120 is an omega-3 fatty acid receptor mediating potent anti-inflammatory and insulin-sensitizing effects

P304

1664-1678

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P31

scholarly article

P356

10.1080/15548627.2017.1345411

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P433

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P478

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P50

Geir Bjørkøy

Jan Kristian Damås

P577

2017-08-18T00:00:00Z

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P698

28820283

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P932

5640206

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