Two overlapping sequence motifs within the polyomavirus enhancer are independently the targets of stimulation by both the tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the Ha-ras oncogene.
about
RREB-1, a novel zinc finger protein, is involved in the differentiation response to Ras in human medullary thyroid carcinomasTranscriptional enhancement of the human gene encoding for a melanoma-associated antigen (ME491) in association with malignant transformationDifferentiation of F9 embryonal carcinoma cells induced by the c-jun and activated c-Ha-ras oncogenes.Synergistic activation of the HTLV1 LTR Ets-responsive region by transcription factors Ets1 and Sp1.Loss of DNA-binding and new transcriptional trans-activation function in polyomavirus large T-antigen with mutation of zinc finger motifDNA-binding and transcriptional activation properties of the EWS-FLI-1 fusion protein resulting from the t(11;22) translocation in Ewing sarcomaInteraction of nuclear factor EF-1A with the polyomavirus enhancer region.Minimal subenhancer requirements for high-level polyomavirus DNA replication: a cell-specific synergy of PEA3 and PEA1 sites.Purification of a mouse nuclear factor that binds to both the A and B cores of the polyomavirus enhancer.PEA1 and PEA3 enhancer elements are primary components of the polyomavirus late transcription initiator element.Genetic analysis of the enhancer requirements for polyomavirus DNA replication in mice.The nuclear protooncogenes c-jun and c-fos as regulators of DNA replication.Activation and suppression of a cryptic promoter in the intron of the human melanoma-associated ME491 antigen gene.A polyomavirus enhancer-binding protein, PEBP5, responsive to 12-O-tetradecanoylphorbol-13-acetate but distinct from AP-1.Extracellular signal-regulated kinase signaling regulates the opposing roles of JUN family transcription factors at ETS/AP-1 sites and in cell migration.The capacity of polyomavirus enhancer binding protein 2alphaB (AML1/Cbfa2) to stimulate polyomavirus DNA replication is related to its affinity for the nuclear matrix.Extra mouse mammary tumor proviruses in DBA/2 mouse lymphomas acquire a selective advantage in lymphocytes by alteration in the U3 region of the long terminal repeat.Loss of responsiveness of an AP1-related factor, PEBP1, to 12-O-tetradecanoylphorbol-13-acetate after transformation of NIH 3T3 cells by the Ha-ras oncogene.Compilation of vertebrate-encoded transcription factorsBinding sites of HeLa cell nuclear proteins on the upstream region of adenovirus type 5 E1A gene.c-Jun stimulates origin-dependent DNA unwinding by polyomavirus large Tantigen.RAS/ERK pathway transcriptional regulation through ETS/AP-1 binding sites.Differential expression of subspecies of polyomavirus and murine leukemia virus enhancer core binding protein, PEBP2, in various hematopoietic cells.
P2860
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P2860
Two overlapping sequence motifs within the polyomavirus enhancer are independently the targets of stimulation by both the tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the Ha-ras oncogene.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
1989年學術文章
@zh
1989年學術文章
@zh-hant
name
Two overlapping sequence motif ...... etate and the Ha-ras oncogene.
@en
type
label
Two overlapping sequence motif ...... etate and the Ha-ras oncogene.
@en
prefLabel
Two overlapping sequence motif ...... etate and the Ha-ras oncogene.
@en
P2093
P2860
P1433
P1476
Two overlapping sequence motif ...... etate and the Ha-ras oncogene.
@en
P2093
P2860
P304
P407
P577
1989-03-01T00:00:00Z