B Cell-Specific Expression of Ataxia-Telangiectasia Mutated Protein Kinase Promotes Chronic Gammaherpesvirus Infection.

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B Cell-Specific Expression of Ataxia-Telangiectasia Mutated Protein Kinase Promotes Chronic Gammaherpesvirus Infection.

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B Cell-Specific Expression of ...... ic Gammaherpesvirus Infection.

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B Cell-Specific Expression of ...... ic Gammaherpesvirus Infection.

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B Cell-Specific Expression of ...... ic Gammaherpesvirus Infection.

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P1476

B Cell-Specific Expression of ...... ic Gammaherpesvirus Infection.

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Eric J Darrah

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Gang Xin

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Joseph M Kulinski

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Laurent P Malherbe

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Stephen B Gauld

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Vera L Tarakanova

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Wadzanai P Mboko

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Weiguo Cui

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P2860

A single ataxia telangiectasia gene with a product similar to PI-3 kinase

Immunoglobulin class switch recombination is impaired in Atm-deficient mice

A gammaherpesvirus Bcl-2 ortholog blocks B cell receptor-mediated apoptosis and promotes the survival of developing B cells in vivo

Gammaherpesvirus-driven plasma cell differentiation regulates virus reactivation from latently infected B lymphocytes.

Antibody-independent control of gamma-herpesvirus latency via B cell induction of anti-viral T cell responses.

Inhibition of NF-kappaB activation in vivo impairs establishment of gammaherpesvirus latency.

Cutaneous granulomatosis and combined immunodeficiency revealing Ataxia-Telangiectasia: a case report

Immunodeficiency and infections in ataxia-telangiectasia.

Epstein-Barr-virus-carrying lymphoma in a patient with ataxia-telangiectasia.

Long-term latent murine Gammaherpesvirus 68 infection is preferentially found within the surface immunoglobulin D-negative subset of splenic B cells in vivo.

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scholarly article

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10.1128/JVI.01103-17

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English

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2017-09-12T00:00:00Z

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28701397

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5599758

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