2-5A accumulates to high levels in interferon-treated, vaccinia virus-infected cells in the absence of any inhibition of virus replication.
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Diverse functions of RNase L and implications in pathologyViral encounters with 2',5'-oligoadenylate synthetase and RNase L during the interferon antiviral responseRegulation of mRNA Translation and Cellular Signaling by Hepatitis C Virus Nonstructural Protein NS5ARNase L inhibitor is induced during human immunodeficiency virus type 1 infection and down regulates the 2-5A/RNase L pathway in human T cells.Viral phosphodiesterases that antagonize double-stranded RNA signaling to RNase L by degrading 2-5AEvasion of antiviral innate immunity by Theiler's virus L* protein through direct inhibition of RNase LHuman 2'-phosphodiesterase localizes to the mitochondrial matrix with a putative function in mitochondrial RNA turnover.Poxvirus decapping enzymes enhance virulence by preventing the accumulation of dsRNA and the induction of innate antiviral responsesEnzyme assays for synthesis and degradation of 2-5As and other 2'-5' oligonucleotides.Reversal of the interferon-sensitive phenotype of a vaccinia virus lacking E3L by expression of the reovirus S4 gene.Modulation of ppp(A2'p)nA-dependent RNase by a temperature-sensitive mutant of vaccinia virusInterferon prevents the generation of spontaneous deletions at the left terminus of vaccinia virus DNA.Characterization of a temperature-sensitive mutant of vaccinia virus reveals a novel function that prevents virus-induced breakdown of RNADouble-stranded RNA-dependent protein kinase and 2-5A system are both activated in interferon-treated, encephalomyocarditis virus-infected HeLa cells.Interferon-mediated, double-stranded RNA-dependent protein kinase is inhibited in extracts from vaccinia virus-infected cellsRole of ribonuclease L in viral pathogen-associated molecular pattern/influenza virus and cigarette smoke-induced inflammation and remodeling.Structural analysis of the human interferon gamma receptor: a small segment of the intracellular domain is specifically required for class I major histocompatibility complex antigen induction and antiviral activity.Novel 2',5'-oligoadenylates synthesized in interferon-treated, vaccinia virus-infected cells.Analogue inhibitor of 2-5A action: effect on the interferon-mediated inhibition of encephalomyocarditis virus replication.A novel mechanism of RNase L inhibition: Theiler's virus L* protein prevents 2-5A from binding to RNase L.
P2860
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P2860
2-5A accumulates to high levels in interferon-treated, vaccinia virus-infected cells in the absence of any inhibition of virus replication.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
2-5A accumulates to high level ...... hibition of virus replication.
@en
type
label
2-5A accumulates to high level ...... hibition of virus replication.
@en
prefLabel
2-5A accumulates to high level ...... hibition of virus replication.
@en
P2093
P2860
P1433
P1476
2-5A accumulates to high level ...... hibition of virus replication.
@en
P2093
P2860
P304
P407
P577
1984-04-01T00:00:00Z