Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells. - Wikidata - wikimedia - TriplyDB

Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

http://www.wikidata.org/entity/Q40141225

about

Three dysregulated miRNAs control kallikrein 10 expression and cell proliferation in ovarian cancer.Identification and validation of genes involved in cervical tumourigenesis.Protein-binding microarray analysis of tumor suppressor AP2α target gene specificity.Genomic instability and copy-number heterogeneity of chromosome 19q, including the kallikrein locus, in ovarian carcinomas.Down-regulation of kallikrein-related peptidase 5 (KLK5) expression in breast cancer patients: a biomarker for the differential diagnosis of breast lesions Expression and function of the kallikrein-related peptidase 6 in the human melanoma microenvironment.Identification of prefoldin amplification (1q23.3-q24.1) in bladder cancer using comparative genomic hybridization (CGH) arrays of urinary DNA microRNAs: a new frontier in kallikrein research.Prognostic value and biological role of the kallikrein-related peptidases in human malignancies.The physiology and pathobiology of human kallikrein-related peptidase 6 (KLK6).Predictions for the future of kallikrein-related peptidases in molecular diagnostics.Kallikrein-related peptidase expression in odontogenic cysts and tumors: An immunohistochemical comparative study.LY6K is a novel molecular target in bladder cancer on basis of integrate genome-wide profiling.Frequent silencing of protocadherin 17, a candidate tumour suppressor for esophageal squamous cell carcinoma.Dysregulation of kallikrein-related peptidases in renal cell carcinoma: potential targets of miRNAs.Activation of hepatocyte growth factor activator zymogen (pro-HGFA) by human kallikrein 1-related peptidases.Impact of cytogenetic and genomic aberrations of the kallikrein locus in ovarian cancer.Downregulated KLK13 expression in bladder cancer highlights tumor aggressiveness and unfavorable patients' prognosis.Effect of Productive Human Papillomavirus 16 Infection on Global Gene Expression in Cervical Epithelium

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Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

http://www.wikidata.org/entity/Q40141225

description

2007 nî lūn-bûn

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name

Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

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Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

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prefLabel

Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells.

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J.1349-7006.2007.00495.X

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Association of KLK5 overexpression with invasiveness of urinary bladder carcinoma cells

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Hitoshi Tsuda

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Johji Inazawa

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Ken-Ichi Kozaki

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Taro Shuin

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Tomoaki Fujioka

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Tsuneharu Miki

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Wataru Obara

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Yasuo Shinoda

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Yoichi Mizutani

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P2860

Tissue-specific expression patterns and fine mapping of the human kallikrein (KLK) locus on proximal 19q13.4

Biochemical characterization of human kallikrein 8 and its possible involvement in the degradation of extracellular matrix proteins

Characterization of the enzymatic activity of human kallikrein 6: Autoactivation, substrate specificity, and regulation by inhibitors

The expanded human kallikrein (KLK) gene family: genomic organisation, tissue-specific expression and potential functions

ADAM23, a possible tumor suppressor gene, is frequently silenced in gastric cancers by homozygous deletion or aberrant promoter hypermethylation

The emerging roles of human tissue kallikreins in cancer

The new human tissue kallikrein gene family: structure, function, and association to disease.

Prognostic value of the human kallikrein gene 15 expression in ovarian cancer.

Frequent silencing of low density lipoprotein receptor-related protein 1B (LRP1B) expression by genetic and epigenetic mechanisms in esophageal squamous cell carcinoma.

Frequent silencing of DBC1 is by genetic or epigenetic mechanisms in non-small cell lung cancers.

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