Zebrafish slc30a10 deficiency revealed a novel compensatory mechanism of Atp2c1 in maintaining manganese homeostasis.
about
Manganese transporter Slc39a14 deficiency revealed its key role in maintaining manganese homeostasis in mice.Hypothyroidism induced by loss of the manganese efflux transporter SLC30A10 may be explained by reduced thyroxine production.Familial manganese-induced neurotoxicity due to mutations in SLC30A10 or SLC39A14.The Essential Element Manganese, Oxidative Stress, and Metabolic Diseases: Links and Interactions.Zebrafish as an Animal Model for Drug Discovery in Parkinson's Disease and Other Movement Disorders: A Systematic Review.Genetic Renal Diseases: The Emerging Role of Zebrafish Models
P2860
Zebrafish slc30a10 deficiency revealed a novel compensatory mechanism of Atp2c1 in maintaining manganese homeostasis.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Zebrafish slc30a10 deficiency ...... taining manganese homeostasis.
@en
type
label
Zebrafish slc30a10 deficiency ...... taining manganese homeostasis.
@en
prefLabel
Zebrafish slc30a10 deficiency ...... taining manganese homeostasis.
@en
P2093
P2860
P1433
P1476
Zebrafish slc30a10 deficiency ...... taining manganese homeostasis.
@en
P2093
Caiyong Chen
Junxia Min
Xiaoli Hong
Yingniang Li
Zhidan Xia
P2860
P304
P356
10.1371/JOURNAL.PGEN.1006892
P50
P577
2017-07-10T00:00:00Z