Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Human Embryonic Stem Cell-Derived Neurons Are Highly Permissive for Varicella-Zoster Virus Lytic Infection.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Arun Venkatesan

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Balaji Jagdish

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Gabsang Lee

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Jeffrey I Cohen

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Labchan Rajbhandari

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Mir A Ali

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Priya Shukla

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Sravya Kurapati

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Yong Jun Kim

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P2860

A comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivation

Functional Coupling with Cardiac Muscle Promotes Maturation of hPSC-Derived Sympathetic Neurons.

Mitogen-activated protein kinases in innate immunity.

Selection and characterization of varicella-zoster virus variants resistant to (R)-9-[4-hydroxy-2-(hydroxymethy)butyl]guanine

Transient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neurons

MAP kinases

Neuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.

Cellular transcriptome analysis reveals differential expression of pro- and antiapoptosis genes by varicella-zoster virus-infected neurons and fibroblasts.

Varicella-zoster virus infection of human dorsal root ganglia in vivo.

Herpes simplex virus ICP27 activation of stress kinases JNK and p38.

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scholarly article

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10.1128/JVI.00640-17

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English

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Tomohiko Sadaoka

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2017-06-21T00:00:00Z

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28637759

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5553188

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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Role of the JNK Pathway in Varicella-Zoster Virus Lytic Infection and Reactivation.

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