Xeroderma pigmentosum group C gene expression is predominantly regulated by promoter hypermethylation and contributes to p53 mutation in lung cancers.
about
A critical re-assessment of DNA repair gene promoter methylation in non-small cell lung carcinomaEpigenetic changes of DNA repair genes in cancerThe clinical value of aberrant epigenetic changes of DNA damage repair genes in human cancerEpigenetic regulation of DNA repair machinery in Helicobacter pylori-induced gastric carcinogenesisSkin cancer in organ transplant recipients: effects of immunosuppressive medications on DNA repairXPC mRNA level may predict relapse in never-smokers with non-small cell lung cancersInter-individual variation in DNA repair capacity: a need for multi-pathway functional assays to promote translational DNA repair researchThe role of Bcl-x(L) protein in nucleotide excision repair-facilitated cell protection against cisplatin-induced apoptosis.Hypoxia-inducible factor-1alpha regulates the expression of nucleotide excision repair proteins in keratinocytes.The role of XPC: implications in cancer and oxidative DNA damageGenetic Evidence for XPC-KRAS Interactions During Lung Cancer DevelopmentXPC inhibits NSCLC cell proliferation and migration by enhancing E-Cadherin expression.Methylation profiling defines an extensive field defect in histologically normal prostate tissues associated with prostate cancerProgrammed genetic instability: a tumor-permissive mechanism for maintaining the evolvability of higher species through methylation-dependent mutation of DNA repair genes in the male germ line.Loss of ARF sensitizes transgenic BRAFV600E mice to UV-induced melanoma via suppression of XPC.MicroRNA-346 facilitates cell growth and metastasis, and suppresses cell apoptosis in human non-small cell lung cancer by regulation of XPC/ERK/Snail/E-cadherin pathway.DNA Repair Gene Expression Levels as Indicators of Breast Cancer in the Breast Cancer Family Registry.Implications of endoplasmic reticulum stress, the unfolded protein response and apoptosis for molecular cancer therapy. Part I: targeting p53, Mdm2, GADD153/CHOP, GRP78/BiP and heat shock proteins.Molecular regulation of UV-induced DNA repair.Histone deacetylases (HDACs) in XPC gene silencing and bladder cancer.Malignant melanoma cells acquire resistance to DNA interstrand cross-linking chemotherapeutics by p53-triggered upregulation of DDB2/XPC-mediated DNA repair.5-Azacytidine facilitates osteogenic gene expression and differentiation of mesenchymal stem cells by alteration in DNA methylation.Epigenetic inactivation of tumour suppressor coding and non-coding genes in human cancer: an update.Clinicopathological significance and prognostic value of Xeroderma pigmentosum complementary group C (XPC) expression in sporadic breast cancer patients.XPC Deficiency Alters Cigarette Smoke DNA Damage Cell Fate and Accelerates Emphysema Development.XPC polymorphisms play a role in tissue-specific carcinogenesis: a meta-analysis.Nucleotide Excision Repair Factor XPC Ameliorates Prognosis by Increasing the Susceptibility of Human Colorectal Cancer to Chemotherapy and Ionizing Radiation
P2860
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P2860
Xeroderma pigmentosum group C gene expression is predominantly regulated by promoter hypermethylation and contributes to p53 mutation in lung cancers.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Xeroderma pigmentosum group C ...... p53 mutation in lung cancers.
@en
type
label
Xeroderma pigmentosum group C ...... p53 mutation in lung cancers.
@en
prefLabel
Xeroderma pigmentosum group C ...... p53 mutation in lung cancers.
@en
P2093
P356
P1433
P1476
Xeroderma pigmentosum group C ...... p53 mutation in lung cancers.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1210284
P407
P577
2007-02-26T00:00:00Z
P5875
P6179
1052634510