Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
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Evidence for the extended phospholipid conformation in membrane fusion and hemifusionNaja naja oxiana Cobra Venom Cytotoxins CTI and CTII Disrupt Mitochondrial Membrane Integrity: Implications for Basic Three-Fingered CytotoxinsEnergetics of intermediates in membrane fusion: comparison of stalk and inverted micellar intermediate mechanismsModel of interaction between a cardiotoxin and dimyristoylphosphatidic acid bilayers determined by solid-state 31P NMR spectroscopy.Morphological responses to calcium-induced interaction of phosphatidylserine-containing vesicles.Genetic ablation of calcium-independent phospholipase A2gamma leads to alterations in mitochondrial lipid metabolism and function resulting in a deficient mitochondrial bioenergetic phenotypeInverted micellar intermediates and the transitions between lamellar, cubic, and inverted hexagonal lipid phases. I. Mechanism of the L alpha----HII phase transitions.The mechanism of lamellar-to-inverted hexagonal phase transitions: a study using temperature-jump cryo-electron microscopyImportance of the hexagonal lipid phase in biological membrane organization.On the correlation between HII phase and the contact-induced destabilization of phosphatidylethanolamine-containing membranes.Intermediates in membrane fusion and bilayer/nonbilayer phase transitions imaged by time-resolved cryo-transmission electron microscopy.Amphiphilic beta-sheet cobra cardiotoxin targets mitochondria and disrupts its network.Observations of membrane fusion in a liposome dispersion: the missing fusion intermediate?Membrane permeabilization induced by sphingosine: effect of negatively charged lipids.Inverted micellar intermediates and the transitions between lamellar, cubic, and inverted hexagonal lipid phases. II. Implications for membrane-membrane interactions and membrane fusion.End-products diacylglycerol and ceramide modulate membrane fusion induced by a phospholipase C/sphingomyelinase from Pseudomonas aeruginosaAcyl chain orientational order in large unilamellar vesicles: comparison with multilamellar liposomes: a 2H and 31P nuclear magnetic resonance study.Pore formation by equinatoxin II, a eukaryotic protein toxin, occurs by induction of nonlamellar lipid structures.Continuous formation of supported cubic and hexagonal mesoporous films by sol–gel dip-coating
P2860
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P2860
Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
@en
type
label
Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
@en
prefLabel
Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
@en
P2860
P1433
P1476
Inverted micellar structures in bilayer membranes. Formation rates and half-lives.
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(84)84164-8
P407
P577
1984-02-01T00:00:00Z