Cellular mechanisms of mainstream cigarette smoke-induced lung epithelial tight junction permeability changes in vitro.
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Lung microbiology and exacerbations in COPDPulmonary epithelial barrier function: some new players and mechanismsAssessment of cigarette smoke particle deposition within the Vitrocell® exposure module using quartz crystal microbalancesPrevalence and impact of active and passive cigarette smoking in acute respiratory distress syndrome.Outgrowth of the bacterial airway microbiome after rhinovirus exacerbation of chronic obstructive pulmonary disease.Phenotypic and genetic aspects of epithelial barrier function in asthmatic patients.Molecular processes that drive cigarette smoke-induced epithelial cell fate of the lung.Systemic inflammation in chronic obstructive pulmonary disease: may adipose tissue play a role? Review of the literature and future perspectives.Hypertonic challenge to porcine vocal folds: effects on epithelial barrier function.Localization of type I interferon receptor limits interferon-induced TLR3 in epithelial cells.Hyaluronan and layilin mediate loss of airway epithelial barrier function induced by cigarette smoke by decreasing E-cadherin.Sidestream smoke exposure increases the susceptibility of airway epithelia to adenoviral infectionThe Rho pathway mediates transition to an alveolar type I cell phenotype during static stretch of alveolar type II cells.Effects of molecular structural variants on serum Krebs von den Lungen-6 levels in sarcoidosis.Maternal smoke exposure decreases mesenchymal proliferation and modulates Rho-GTPase-dependent actin cytoskeletal signaling in fetal lungs.Utility of urinary Clara cell protein (CC16) to demonstrate increased lung epithelial permeability in non-smokers exposed to outdoor secondhand smokeAir pollution and respiratory viral infection.Pulmonary epithelium, cigarette smoke, and chronic obstructive pulmonary disease.Cigarette smoke impairs clearance of apoptotic cells through oxidant-dependent activation of RhoACigarette smoke extract-induced p120-mediated NF-κB activation in human epithelial cells is dependent on the RhoA/ROCK pathwayNatural killer T cells are not the predominant T cell in asthma and likely modulate, not cause, asthma.The airway epithelium in asthma.Airway responses towards allergens - from the airway epithelium to T cells.Epithelial barrier function and immunity in asthma.Methodological considerations when conducting in vitro, air-liquid interface exposures to engineered nanoparticle aerosols.Aquaporin 5 regulates cigarette smoke induced emphysema by modulating barrier and immune properties of the epithelium.Cigarette smoke-induced disruption of bronchial epithelial tight junctions is prevented by transforming growth factor-β.Analytical in vitro approach for studying cyto- and genotoxic effects of particulate airborne material.Direct exposure at the air-liquid interface: evaluation of an in vitro approach for simulating inhalation of airborne substances.Up-Regulation of Claudin-6 in the Distal Lung Impacts Secondhand Smoke-Induced InflammationCytokines induce tight junction disassembly in airway cells via an EGFR-dependent MAPK/ERK1/2-pathway.Rho kinases regulate corneal epithelial wound healing.Risk factors for community-acquired pneumonia in pre-school-aged children.A systems biology approach reveals the dose- and time-dependent effect of primary human airway epithelium tissue culture after exposure to cigarette smoke in vitroCigarette smoking reprograms apical junctional complex molecular architecture in the human airway epithelium in vivo.Primary human sinonasal epithelial cell culture model for topical drug delivery in patients with chronic rhinosinusitis with nasal polyposis.Correlative ultrastructural investigations of airway epithelium following experimental exposure to defined air pollutants and lifestyle exposure to tobacco smoke.Evidence for involvement of ROCK signaling in bradykinin-induced increase in murine blood-tumor barrier permeability.Chronic Inhalation of E-Cigarette Vapor Containing Nicotine Disrupts Airway Barrier Function and Induces Systemic Inflammation and Multi-Organ Fibrosis in Mice.Application of dosimetry tools for the assessment of e-cigarette aerosol and cigarette smoke generated on two different in vitro exposure systems.
P2860
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P2860
Cellular mechanisms of mainstream cigarette smoke-induced lung epithelial tight junction permeability changes in vitro.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Cellular mechanisms of mainstr ...... permeability changes in vitro.
@en
type
label
Cellular mechanisms of mainstr ...... permeability changes in vitro.
@en
prefLabel
Cellular mechanisms of mainstr ...... permeability changes in vitro.
@en
P2093
P2860
P1476
Cellular mechanisms of mainstr ...... permeability changes in vitro.
@en
P2093
Chris Beenhouwer
Cindy Knall
Dorian S Olivera
James Aden
Susan E Boggs
P2860
P356
10.1080/08958370600985768
P577
2007-01-01T00:00:00Z