Gonococcal porin IB activates NF-kappaB in human urethral epithelium and increases the expression of host antiapoptotic factors.
about
Modulation of tumor necrosis factor by microbial pathogens.Neisseria gonorrhoeae activates the proteinase cathepsin B to mediate the signaling activities of the NLRP3 and ASC-containing inflammasomeExpression capable library for studies of Neisseria gonorrhoeae, version 1.0.Crystallographic analysis of Neisseria meningitidis PorB extracellular loops potentially implicated in TLR2 recognitionNeisseriae internalization by epithelial cells is enhanced by TLR2 stimulationPhagocytosis of Staphylococcus aureus by macrophages exerts cytoprotective effects manifested by the upregulation of antiapoptotic factors.Meningococcal porin PorB prevents cellular apoptosis in a toll-like receptor 2- and NF-kappaB-independent mannerNleH effectors interact with Bax inhibitor-1 to block apoptosis during enteropathogenic Escherichia coli infection.Biofilm Formation by Neisseria gonorrhoeae.Infection of human fallopian tube epithelial cells with Neisseria gonorrhoeae protects cells from tumor necrosis factor alpha-induced apoptosis.Neisseria gonorrhoeae-mediated inhibition of apoptotic signalling in polymorphonuclear leukocytes.Shigella flexneri inhibits staurosporine-induced apoptosis in epithelial cells.Infection with Anaplasma phagocytophilum activates the phosphatidylinositol 3-Kinase/Akt and NF-κB survival pathways in neutrophil granulocytes.The mechanism of Jurkat cells apoptosis induced by Aggregatibacter actinomycetemcomitans cytolethal distending toxin.Extracellular signal-regulated kinase activation by Neisseria gonorrhoeae downregulates epithelial cell proapoptotic proteins Bad and Bim.Phenotypic and genotypic analyses of Neisseria gonorrhoeae isolates that express frequently recovered PorB PIA variable region types suggest that certain P1a porin sequences confer a selective advantage for urogenital tract infectionStaying alive: bacterial inhibition of apoptosis during infection.Neisseria gonorrhoeae infection protects human endocervical epithelial cells from apoptosis via expression of host antiapoptotic proteins.Structure-function studies of the Neisseria gonorrhoeae major outer membrane porinSaturating mutagenesis of an essential gene: a majority of the Neisseria gonorrhoeae major outer membrane porin (PorB) is mutable.Neisseria gonorrhoeae survives within and modulates apoptosis and inflammatory cytokine production of human macrophagesActivation of NF-κB by Neisseria gonorrhoeae is associated with microcolony formation and type IV pilus retraction.Neisseria meningitidis Lacking the Major Porins PorA and PorB Is Viable and Modulates Apoptosis and the Oxidative Burst of Neutrophils.Neisseria gonorrhoeae delays the onset of apoptosis in polymorphonuclear leukocytes.Anti-Inflammatory Effect of Feiyangchangweiyan Capsule on Rat Pelvic Inflammatory Disease through JNK/NF-κB Pathway.
P2860
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P2860
Gonococcal porin IB activates NF-kappaB in human urethral epithelium and increases the expression of host antiapoptotic factors.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Gonococcal porin IB activates ...... of host antiapoptotic factors.
@en
type
label
Gonococcal porin IB activates ...... of host antiapoptotic factors.
@en
prefLabel
Gonococcal porin IB activates ...... of host antiapoptotic factors.
@en
P2860
P1476
Gonococcal porin IB activates ...... of host antiapoptotic factors.
@en
P2093
Matthew J Binnicker
Richard D Williams
P2860
P304
P356
10.1128/IAI.72.11.6408-6417.2004
P407
P577
2004-11-01T00:00:00Z