Deletion of GSTA4-4 results in increased mitochondrial post-translational modification of proteins by reactive aldehydes following chronic ethanol consumption in mice.
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Aberrant post-translational protein modifications in the pathogenesis of alcohol-induced liver injuryLiver-Specific Deletion of Phosphatase and Tensin Homolog Deleted on Chromosome 10 Significantly Ameliorates Chronic EtOH-Induced Increases in Hepatocellular DamageOxidative stress and lipotoxicityAlcoholic Liver Disease: Pathogenesis and Current Management.Determination of Protein Haptenation by Chemical Sensitisers within the Complexity of the Human Skin Proteome.Dysregulation of antioxidant responses in patients diagnosed with concomitant Primary Sclerosing Cholangitis/Inflammatory Bowel Disease.Differential carbonylation of proteins in end-stage human fatty and nonfatty NASH.Transcriptional profiling of liver tissues in chicken embryo at day 16 and 20 using RNA sequencing reveals differential antioxidant enzyme activity.Glutathione S-Transferase Alpha 4 Prevents Dopamine Neurodegeneration in a Rat Alpha-Synuclein Model of Parkinson's Disease.Elevated Nrf-2 responses are insufficient to mitigate protein carbonylation in hepatospecific PTEN deletion mice.
P2860
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P2860
Deletion of GSTA4-4 results in increased mitochondrial post-translational modification of proteins by reactive aldehydes following chronic ethanol consumption in mice.
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name
Deletion of GSTA4-4 results in ...... c ethanol consumption in mice.
@en
type
label
Deletion of GSTA4-4 results in ...... c ethanol consumption in mice.
@en
prefLabel
Deletion of GSTA4-4 results in ...... c ethanol consumption in mice.
@en
P2093
P2860
P1433
P1476
Deletion of GSTA4-4 results in ...... c ethanol consumption in mice.
@en
P2093
Alisabeth H Shearn
Bridgette Engi
Colin T Shearn
David J Orlicky
Dennis R Petersen
James J Galligan
Kelly E Mercer
Laura M Saba
Martin J Ronis
Piotr Zimniak
P2860
P356
10.1016/J.REDOX.2015.11.013
P577
2015-11-27T00:00:00Z