Nontypable group B streptococci isolated from human sources.
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Invasion of brain microvascular endothelial cells by group B streptococciCharacterization of Fibrinogen Binding by Glycoproteins Srr1 and Srr2 of Streptococcus agalactiaeImmunochemistry of capsular type polysaccharide and virulence properties of type VI Streptococcus agalactiae (group B streptococci).Sudden increase in isolation of group B streptococci, serotype V, is not due to emergence of a new pulsed-field gel electrophoresis typeGroup B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivoClonal relationship between U.S. and French serotype V group B streptococcus isolates.Group B streptococcal serine-rich repeat proteins promote interaction with fibrinogen and vaginal colonizationGroup B Streptococcus interactions with human meningeal cells and astrocytes in vitro.Abundance of the delta subunit of RNA polymerase is linked to the virulence of Streptococcus agalactiaeβ-Hemolysin/cytolysin of Group B Streptococcus enhances host inflammation but is dispensable for establishment of urinary tract infection.Role of the serine-rich surface glycoprotein Srr1 of Streptococcus agalactiae in the pathogenesis of infective endocarditis.Complete Genome Sequence of Streptococcus agalactiae CNCTC 10/84, a Hypervirulent Sequence Type 26 Strain.The role of autophagy during group B Streptococcus infection of blood-brain barrier endotheliumA serotype V clone is predominant among erythromycin-resistant Streptococcus agalactiae isolates in a southwestern region of Germany.Streptococcus agalactiae infection in zebrafish larvaeGroup B streptococcal beta-hemolysin expression is associated with injury of lung epithelial cells.Serine-rich repeat proteins and pili promote Streptococcus agalactiae colonization of the vaginal tract.Sequence type 1 group B Streptococcus, an emerging cause of invasive disease in adults, evolves by small genetic changes.Group B streptococcal infection and activation of human astrocytes.Bacterial induction of Snail1 contributes to blood-brain barrier disruption.Identification of CiaR Regulated Genes That Promote Group B Streptococcal Virulence and Interaction with Brain Endothelial CellsComplement and antibody participation in opsonophagocytosis of type IV and V group B streptococci.Regulation of cytotoxin expression by converging eukaryotic-type and two-component signalling mechanisms in Streptococcus agalactiae.Screening of type Ia and Ib Streptococcus agalactiae strains with high sialic acid levels by determination of susceptibility to tetracyclinesA novel C5a-derived immunobiotic peptide reduces Streptococcus agalactiae colonization through targeted bacterial killing.Worldwide distribution of two new serotypes of group B streptococci: type IV and provisional type V.A group B streptococcal pilus protein promotes phagocyte resistance and systemic virulence.New serotypes of group B streptococci isolated from human sources.The group B streptococcal serine-rich repeat 1 glycoprotein mediates penetration of the blood-brain barrier
P2860
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P2860
Nontypable group B streptococci isolated from human sources.
description
1977 nî lūn-bûn
@nan
1977年の論文
@ja
1977年論文
@yue
1977年論文
@zh-hant
1977年論文
@zh-hk
1977年論文
@zh-mo
1977年論文
@zh-tw
1977年论文
@wuu
1977年论文
@zh
1977年论文
@zh-cn
name
Nontypable group B streptococci isolated from human sources.
@en
type
label
Nontypable group B streptococci isolated from human sources.
@en
prefLabel
Nontypable group B streptococci isolated from human sources.
@en
P2860
P1476
Nontypable group B streptococci isolated from human sources.
@en
P2093
Wilkinson HW
P2860
P304
P407
P577
1977-08-01T00:00:00Z