Differential susceptibilities of mice genomically deleted of CD4 and CD8 to infections with Trypanosoma cruzi or Trypanosoma brucei.
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Immunization with cDNA expressed by amastigotes of Trypanosoma cruzi elicits protective immune response against experimental infectionGenes from Chagas susceptibility loci that are differentially expressed in T. cruzi-resistant mice are candidates accounting for impaired immunity.NFATc1 mediates Toll-like receptor-independent innate immune responses during Trypanosoma cruzi infection.The liver plays a major role in clearance and destruction of blood trypomastigotes in Trypanosoma cruzi chronically infected mice.Prophylactic efficacy of TcVac2 against Trypanosoma cruzi in mice.A synthetic peptide from Trypanosoma cruzi mucin-like associated surface protein as candidate for a vaccine against Chagas diseaseTrypanosoma cruzi posttranscriptionally up-regulates and exploits cellular FLIP for inhibition of death-inducing signal.Predominance of CD4 Th1 and CD8 Tc1 cells revealed by characterization of the cellular immune response generated by immunization with a DNA vaccine containing a Trypanosoma cruzi gene.Immunization of mice with a TolA-like surface protein of Trypanosoma cruzi generates CD4(+) T-cell-dependent parasiticidal activity.Involvement of CD4(+) Th1 cells in systemic immunity protective against primary and secondary challenges with Trypanosoma cruzi.The SA85-1.1 protein of the Trypanosoma cruzi trans-sialidase superfamily is a dominant T-cell antigenDNA sequences encoding CD4+ and CD8+ T-cell epitopes are important for efficient protective immunity induced by DNA vaccination with a Trypanosoma cruzi geneReal-time PCR in HIV/Trypanosoma cruzi coinfection with and without Chagas disease reactivation: association with HIV viral load and CD4 level.During Trypanosoma cruzi infection CD1d-restricted NK T cells limit parasitemia and augment the antibody response to a glycophosphoinositol-modified surface proteinInducible nitric oxide synthase is not essential for control of Trypanosoma cruzi infection in mice.Chronic human infection with Trypanosoma cruzi drives CD4+ T cells to immune senescenceLow frequency of circulating CD8+ T stem cell memory cells in chronic chagasic patients with severe forms of the disease.Humoral and cellular immune responses to Trypanosoma cruzi-derived paraflagellar rod proteins in patients with Chagas' disease.Deficiency of antigen-specific B cells results in decreased Trypanosoma cruzi systemic but not mucosal immunity due to CD8 T cell exhaustion.Pure paraflagellar rod protein protects mice against Trypanosoma cruzi infectionRegulation of Trypanosoma cruzi infection in mice by gamma interferon and interleukin 10: role of NK cellsInterleukin-12-mediated resistance to Trypanosoma cruzi is dependent on tumor necrosis factor alpha and gamma interferon.Importance of beta2-microglobulin in murine resistance to mucosal and systemic candidiasis.A DNA vaccine encoding for TcSSP4 induces protection against acute and chronic infection in experimental Chagas diseaseIL-10 limits parasite burden and protects against fatal myocarditis in a mouse model of Trypanosoma cruzi infection.NK-, NKT- and CD8-Derived IFNγ Drives Myeloid Cell Activation and Erythrophagocytosis, Resulting in Trypanosomosis-Associated Acute AnemiaLimited role for CD4+ T-cell help in the initial priming of Trypanosoma cruzi-specific CD8+ T cellsPromiscuous Recognition of a Trypanosoma cruzi CD8+ T Cell Epitope among HLA-A2, HLA-A24 and HLA-A1 Supertypes in Chagasic Patients.Previously unrecognized vaccine candidates control Trypanosoma cruzi infection and immunopathology in miceApoptotic CD8 T-lymphocytes disable macrophage-mediated immunity to Trypanosoma cruzi infection.Chagas' disease: an update on immune mechanisms and therapeutic strategiesInfection with Trypanosoma cruzi selectively upregulates B7-2 molecules on macrophages and enhances their costimulatory activity.Immunodepression induced by Trypanosoma cruzi and mouse hepatitis virus type 3 is associated with thymus apoptosis.The gene for a T lymphocyte triggering factor from African trypanosomes.Type 1 immunity provides both optimal mucosal and systemic protection against a mucosally invasive, intracellular pathogen.Absence of CD4+ T lymphocytes, CD8+ T lymphocytes, or B lymphocytes has different effects on the efficacy of posaconazole and benznidazole in treatment of experimental acute Trypanosoma cruzi infection.Gene gun-mediated delivery of an interleukin-12 expression plasmid protects against infections with the intracellular protozoan parasites Leishmania major and Trypanosoma cruzi in mice.Most parasite-specific CD8+ cells in Trypanosoma cruzi-infected chronic mice are down-regulated for T-cell receptor-alphabeta and CD8 molecules.Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis.Differential CD86 and CD40 co-stimulatory molecules and cytokine expression pattern induced by Trypanosoma cruzi in APCs from resistant or susceptible mice
P2860
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P2860
Differential susceptibilities of mice genomically deleted of CD4 and CD8 to infections with Trypanosoma cruzi or Trypanosoma brucei.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Differential susceptibilities ...... a cruzi or Trypanosoma brucei.
@en
type
label
Differential susceptibilities ...... a cruzi or Trypanosoma brucei.
@en
prefLabel
Differential susceptibilities ...... a cruzi or Trypanosoma brucei.
@en
P2093
P2860
P1476
Differential susceptibilities ...... ma cruzi or Trypanosoma brucei
@en
P2093
P2860
P304
P407
P577
1993-12-01T00:00:00Z