Effects of ATPase inhibitors on the response of HeLa cells to Helicobacter pylori vacuolating toxin.
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Helicobacter pyloriInstrumental Role of Helicobacter pylori γ-Glutamyl Transpeptidase in VacA-Dependent Vacuolation in Gastric Epithelial Cells"Non-Toxic" Proteins of the Botulinum Toxin Complex Exert In-vivo ToxicityThe vacuolating toxin from Helicobacter pylori forms hexameric pores in lipid bilayers at low pH.Mimicry of a host anion channel by a Helicobacter pylori pore-forming toxin.Helicobacter pylori VacA toxin promotes bacterial intracellular survival in gastric epithelial cells.Cloning and characterization of a novel membrane-associated antigenic protein of Helicobacter pylori.Molecular analysis of Culex quinquefasciatus larvae responses to Lysinibacillus sphaericus Bin toxin.Effects of Helicobacter pylori vacuolating cytotoxin on primary cultures of human gastric epithelial cellsVesicles containing Chlamydia trachomatis serovar L2 remain above pH 6 within HEC-1B cells.Binding and internalization of the Helicobacter pylori vacuolating cytotoxin by epithelial cells.Cellular vacuoles induced by Helicobacter pylori originate from late endosomal compartments.Salmonella induces the formation of filamentous structures containing lysosomal membrane glycoproteins in epithelial cells.Clustering and redistribution of late endocytic compartments in response to Helicobacter pylori vacuolating toxin.Cytotoxic action of Serratia marcescens hemolysin on human epithelial cells.Selective increase of the permeability of polarized epithelial cell monolayers by Helicobacter pylori vacuolating toxin.Cytotoxin production by Helicobacter pylori from patients with upper gastrointestinal tract diseases.Helicobacter pylori vacuolating toxin forms anion-selective channels in planar lipid bilayers: possible implications for the mechanism of cellular vacuolation.Determinants of pH-dependent modulation of translocation in dermonecrotic G-protein-deamidating toxins.Gene structure of the Helicobacter pylori cytotoxin and evidence of its key role in gastric disease.The small GTP binding protein rab7 is essential for cellular vacuolation induced by Helicobacter pylori cytotoxin.A Tale of Two Toxins: Helicobacter Pylori CagA and VacA Modulate Host Pathways that Impact Disease.
P2860
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P2860
Effects of ATPase inhibitors on the response of HeLa cells to Helicobacter pylori vacuolating toxin.
description
1993 nî lūn-bûn
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1993年の論文
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1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
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1993年论文
@wuu
1993年论文
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1993年论文
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name
Effects of ATPase inhibitors o ...... cter pylori vacuolating toxin.
@en
type
label
Effects of ATPase inhibitors o ...... cter pylori vacuolating toxin.
@en
prefLabel
Effects of ATPase inhibitors o ...... cter pylori vacuolating toxin.
@en
P2093
P2860
P1476
Effects of ATPase inhibitors o ...... acter pylori vacuolating toxin
@en
P2093
P2860
P304
P407
P577
1993-04-01T00:00:00Z