Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
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HDL in sepsis - risk factor and therapeutic approachHigh-density Lipoproteins and Apolipoprotein A-I: Potential New Players in the Prevention and Treatment of Lung DiseaseGut microbiota and immune crosstalk in metabolic diseaseMetagenome and metabolism: the tissue microbiota hypothesis.Serum lipoproteins attenuate macrophage activation and Toll-Like Receptor stimulation by bacterial lipoproteins.Identification of haptoglobin and apolipoprotein A-I as biomarkers for high altitude pulmonary edema.Apolipoprotein A-I, A-II, and H mRNA and protein accumulation sites in the developing lung in late gestationThe lipid-bound apolipoprotein A-I cysteine mutant (N74C) inhibits the activation of NF-κB, JNK and p38 in endotoxemic mice and RAW264.7 cells.Anti-inflammatory mechanisms of apolipoprotein A-I mimetic peptide in acute respiratory distress syndrome secondary to sepsis.Proteomic analyses of host and pathogen responses during bovine mastitis.Apolipoprotein A-I attenuates ovalbumin-induced neutrophilic airway inflammation via a granulocyte colony-stimulating factor-dependent mechanism.Emerging roles for cholesterol and lipoproteins in lung disease.Apolipoproteins and apolipoprotein mimetic peptides modulate phagocyte trafficking through chemotactic activityCarboxyl-Terminal Cleavage of Apolipoprotein A-I by Human Mast Cell Chymase Impairs Its Anti-Inflammatory Properties.The apolipoprotein m-sphingosine-1-phosphate axis: biological relevance in lipoprotein metabolism, lipid disorders and atherosclerosisDifferential plasma proteome analysis in patients with high-altitude pulmonary edema at the acute and recovery phases.Emerging Roles of Apolipoprotein E and Apolipoprotein A-I in the Pathogenesis and Treatment of Lung Disease.Contribution of the C-terminal end of apolipoprotein AI to neutralization of lipopolysaccharide endotoxic effect.[Gut microbiota and immune crosstalk in metabolic disease].A novel fibrinogenase from Agkistrodon acutus venom protects against LPS-induced endotoxemia via regulating NF-κB pathway.Effect of lipid-bound apoA-I cysteine mutants on lipopolysaccharide-induced endotoxemia in mice.Apolipoproteins inhibit the innate immunity activated by necrotic cells or bacterial endotoxin.L-4F inhibits lipopolysaccharide-mediated activation of primary human neutrophils.Role of lung apolipoprotein A-I in idiopathic pulmonary fibrosis: antiinflammatory and antifibrotic effect on experimental lung injury and fibrosis.Efficacy of specific IgY for treatment of lipopolysaccharide-induced endotoxemia using a mouse model.Low serum high-density lipoprotein cholesterol in childhood is associated with adolescent asthma
P2860
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P2860
Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
@en
type
label
Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
@en
prefLabel
Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
@en
P2093
P1433
P1476
Beneficial effects of ApoA-I on LPS-induced acute lung injury and endotoxemia in mice.
@en
P2093
P304
P356
10.1016/J.LFS.2006.02.011
P407
P577
2006-03-06T00:00:00Z