Hyaluronan is not a ligand but a regulator of toll-like receptor signaling in mesangial cells: role of extracellular matrix in innate immunity.
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Lubricin/Proteoglycan 4 binds to and regulates the activity of Toll-Like Receptors In Vitro.Hyaluronan fragments as mediators of inflammation in allergic pulmonary diseaseAn update on the role of the inflammasomes in the pathogenesis of kidney diseases.A Modified Glycosaminoglycan, GM-0111, Inhibits Molecular Signaling Involved in Periodontitis.Endotoxin free hyaluronan and hyaluronan fragments do not stimulate TNF-α, interleukin-12 or upregulate co-stimulatory molecules in dendritic cells or macrophages.Beyond tissue injury-damage-associated molecular patterns, toll-like receptors, and inflammasomes also drive regeneration and fibrosis.The where, when, how, and why of hyaluronan binding by immune cells.Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity.Synovial fibroblasts in 2017.NFκB activation in differentiating glioblastoma stem-like cells is promoted by hyaluronic acid signaling through TLR4.
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P2860
Hyaluronan is not a ligand but a regulator of toll-like receptor signaling in mesangial cells: role of extracellular matrix in innate immunity.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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name
Hyaluronan is not a ligand but ...... lar matrix in innate immunity.
@en
type
label
Hyaluronan is not a ligand but ...... lar matrix in innate immunity.
@en
prefLabel
Hyaluronan is not a ligand but ...... lar matrix in innate immunity.
@en
P2860
P356
P1433
P1476
Hyaluronan is not a ligand but ...... ular matrix in innate immunity
@en
P2093
Hans-Joachim Anders
Rainer Ebid
P2860
P304
P356
10.1155/2014/714081
P577
2014-01-21T00:00:00Z