Minimal contribution of desmethyl-gefitinib, the major human plasma metabolite of gefitinib, to epidermal growth factor receptor (EGFR)-mediated tumour growth inhibition.
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Functional characterization of wild-type and 24 CYP2D6 allelic variants on gefitinib metabolism in vitro.Metabolites: have we MIST out the importance of structure and physicochemistry?Clinical pharmacokinetics of tyrosine kinase inhibitors: focus on 4-anilinoquinazolines.Metabolism of the EGFR tyrosin kinase inhibitor gefitinib by cytochrome P450 1A1 enzyme in EGFR-wild type non small cell lung cancer cell lines.Inhibition of SN-38 glucuronidation by gefitinib and its metabolite.Gefitinib-phenytoin interaction is not correlated with the C-erythromycin breath test in healthy male volunteersEffects of polymorphisms in CYP2D6 and ABC transporters and side effects induced by gefitinib on the pharmacokinetics of the gefitinib metabolite, O-desmethyl gefitinib.Genetic Association of Drug Response to Erlotinib in Chinese Advanced Non-small Cell Lung Cancer Patients.
P2860
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P2860
Minimal contribution of desmethyl-gefitinib, the major human plasma metabolite of gefitinib, to epidermal growth factor receptor (EGFR)-mediated tumour growth inhibition.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Minimal contribution of desmet ...... ated tumour growth inhibition.
@en
type
label
Minimal contribution of desmet ...... ated tumour growth inhibition.
@en
prefLabel
Minimal contribution of desmet ...... ated tumour growth inhibition.
@en
P2093
P2860
P1433
P1476
Minimal contribution of desmet ...... iated tumour growth inhibition
@en
P2093
D McKillop
M Hutchison
M P Spence
P2860
P356
10.1080/00498250500523253
P407
P50
P577
2006-01-01T00:00:00Z